Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

The CD4-Th1 model for multiple sclerosis: a critical [correction of crucial] re-appraisal.

Trends in immunology - 1 Mar 2004

Lassmann Hans, Ransohoff Richard M

Abstract excerpt

No abstract is available from the source.

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Autoimmune Diseases of the Nervous System
Share to CD4-Positive T-Lymphocytes
Share to CD8-Positive T-Lymphocytes
Share to Encephalomyelitis, Autoimmune, Experimental

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Models, Immunological

Share to Multiple Sclerosis

Share to Nerve Degeneration

Share to Th1 Cells

Source

Identifiers

PMID: 15036040
DOI: 10.1016/j.it.2004.01.007

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 3 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

The CD4-Th1 model for multiple sclerosis: a critical [correction of crucial] re-appraisal.PMID 15036040Read ↗

Multiple sclerosis relapse phenotype is an important, neglected, determinant of disease outcome - YES.Semantic proximity Read ↗

Multiple sclerosis relapse phenotype is an important, neglected determinant of disease-outcome: Commentary.Semantic proximity Read ↗

Multiple sclerosis relapse phenotype is an important, neglected, determinant of disease outcome - NO.Semantic proximity Read ↗

[Multiple sclerosis: when to think of it? How to proceed?].Semantic proximity Read ↗

Which immune cells matter? The immunopathogenesis of multiple sclerosis.Semantic proximity Read ↗

An update on multiple sclerosis.Semantic proximity Read ↗

Multiple sclerosis: immunopathogenesis and controversies in defining the causeSemantic proximity Read ↗

Multiple Sclerosis Re-Examined: Essential and Emerging Clinical Concepts.Semantic proximity Read ↗

[Background and trigger factors for neuroimmunological diseases].Semantic proximity Read ↗

EAE: imperfect but useful models of multiple sclerosisSemantic proximity Read ↗

T cells and microglia as drivers of multiple sclerosis pathology.Semantic proximity Read ↗

IMMUNOLOGY OF MULTIPLE SCLEROSISSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.