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Article

Imprinting of the MEA Polycomb gene is controlled by antagonism between MET1 methyltransferase and DME glycosylase.

Developmental cell - 1 Dec 2003

Xiao Wenyan, Gehring Mary, Choi Yeonhee, Margossian Linda, Pu Hong, Harada John J, Goldberg Robert B, Pennell Roger I, Fischer Robert L

Abstract excerpt

The MEA Polycomb gene is imprinted in the Arabidopsis endosperm. DME DNA glycosylase activates maternal MEA allele expression in the central cell of the female gametophyte, the progenitor of the endosperm. Maternal mutant dme or mea alleles result in seed abortion. We identified mutations that suppress dme seed abortion and found that they reside in the MET1 methyltransferase gene, which maintains cytosine...

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

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Identifiers

PMID: 14667411
DOI: 10.1016/s1534-5807(03)00361-7

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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Imprinting of the MEA Polycomb gene is controlled by antagonism between MET1 methyltransferase and DME glycosylase.PMID 14667411Read ↗

DEMETER DNA glycosylase establishes MEDEA polycomb gene self-imprinting by allele-specific demethylation.Semantic proximity Read ↗

The Polycomb group protein MEDEA and the DNA methyltransferase MET1 interact to repress autonomous endosperm development in Arabidopsis.Semantic proximity Read ↗

DEMETER, a DNA glycosylase domain protein, is required for endosperm gene imprinting and seed viability in arabidopsis.Semantic proximity Read ↗

Imprinting of the MEDEA polycomb gene in the Arabidopsis endosperm.Semantic proximity Read ↗

Maintenance of genomic imprinting at the Arabidopsis medea locus requires zygotic DDM1 activity.Semantic proximity Read ↗

Mutations in the FIE and MEA genes that encode interacting polycomb proteins cause parent-of-origin effects on seed development by distinct mechanisms.Semantic proximity Read ↗

Regulation of imprinted gene expression in Arabidopsis endosperm.Semantic proximity Read ↗

Dynamic regulatory interactions of Polycomb group genes: MEDEA autoregulation is required for imprinted gene expression in Arabidopsis.Semantic proximity Read ↗

Maternal control of embryogenesis by MEDEA, a polycomb group gene in Arabidopsis.Semantic proximity Read ↗

One-way control of FWA imprinting in Arabidopsis endosperm by DNA methylation.Semantic proximity Read ↗

Maintenance of DNA methylation during the Arabidopsis life cycle is essential for parental imprinting.Semantic proximity Read ↗

Imprinting of the polycomb group gene MEDEA serves as a ploidy sensor in Arabidopsis.Semantic proximity Read ↗

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