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Article

Association of eNOS Glu298Asp polymorphism with end-stage renal disease.

Hypertension (Dallas, Tex. : 1979) - 1 Oct 2002

Noiri Eisei, Satoh Hiroaki, Taguchi Jun-ichi, Brodsky Sergey V, Nakao Akihide, Ogawa Yumiko, Nishijima Satomi, Yokomizo Takehiko, Tokunaga Katsushi, Fujita Toshiro

Abstract excerpt

Nitric oxide (NO) derived from endothelial cells is profoundly related to the maintenance of physiological vascular tone. Impairment of endothelial NO generation brought about by gene polymorphism is considered the major deterioration factor for progressive renal disease, including diabetic nephropathy. The present study aimed to elucidate the Glu298Asp polymorphism of endothelial NO synthase (eNOS) in patients...

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PMID: 12364359
DOI: 10.1161/01.hyp.0000033974.57407.82

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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