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Diallyl Disulfide (DADS) Induces the Antitumorigenic NSAID-Activated Gene (NAG-1) by a p53-Dependent Mechanism in Human Colorectal HCT 116 Cells

1 Apr 2002

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

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PMID: 11925476
DOI: 10.1093/jn/132.4.773

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Related research

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Diallyl Disulfide (DADS) Induces the Antitumorigenic NSAID-Activated Gene (NAG-1) by a p53-Dependent Mechanism in Human Colorectal HCT 116 CellsPMID 11925476Read ↗

Diallyl disulfide induces reversible G2/M phase arrest on a p53-independent mechanism in human colon cancer HCT-116 cellsSemantic proximity Read ↗

Expression of NSAID-activated gene-1 by EGCG in head and neck cancer: involvement of ATM-dependent p53 expressionSemantic proximity Read ↗

NSAID Activated Gene (NAG-1), a Modulator of TumorigenesisSemantic proximity Read ↗

Nonsteroidal anti-inflammatory drug-activated gene (NAG-1) is induced by genistein through the expression of p53 in colorectal cancer cells.Semantic proximity Read ↗

Diallyl disulfide causes caspase-dependent apoptosis in human cancer cells through a Bax-triggered mitochondrial pathwaySemantic proximity Read ↗

Diallyl sulfide, diallyl disulfide and diallyl trisulfide affect drug resistant gene expression in colo 205 human colon cancer cells in vitro and in vivoSemantic proximity Read ↗

A novel anticancer effect of Astragalus saponins: Transcriptional activation of NSAID‐activated geneSemantic proximity Read ↗

Diallyl disulfide induces Ca2+ mobilization in human colon cancer cell line SW480.Semantic proximity Read ↗

Aaptamines from the Marine Sponge<i>Aaptos</i>sp. Display Anticancer Activities in Human Cancer Cell Lines and Modulate AP-1-, NF-<i>κ</i>B-, and p53-Dependent Transcriptional Activity in Mouse JB6 Cl41 CellsSemantic proximity Read ↗

Reversion of P-glycoprotein-mediated multidrug resistance by diallyl trisulfide in a human osteosarcoma cell lineSemantic proximity Read ↗

Molecular cloning and characterization of human nonsteroidal anti-inflammatory drug-activated gene promoter. Basal transcription is mediated by Sp1 and Sp3.Semantic proximity Read ↗

10-formyltetrahydrofolate dehydrogenase-induced c-Jun-NH2-kinase pathways diverge at the c-Jun-NH2-kinase substrate level in cells with different p53 status.Semantic proximity Read ↗

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