Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Upregulation of the HLH Id gene family in neural progenitors and glial cells of the rat spinal cord following contusion injury.

Journal of neuroscience research - 15 Dec 2001

Tzeng S F, Bresnahan J C, Beattie M S, de Vellis J

Abstract excerpt

Spinal cord injury (SCI) leads to a complex sequence of cellular responses, including astrocyte activation, oligodendrocyte death, and ependymal cell proliferation. Inhibitors of DNA binding (Id1, Id2, Id3) belong to a helix-loop-helix (HLH) gene family. Id genes have been implicated in playing a vital role in the proliferation of many cell types, including astrocytes and myoblasts. In the present study, the...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Glial Fibrillary Acidic Protein
Share to Helix-Loop-Helix Motifs

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Intermediate Filament Proteins

Share to Nestin

Share to Oligodendroglia

Share to Rats, Long-Evans

Share to S100 Proteins

Share to Spinal Cord Injuries

Source

Identifiers

PMID: 11746449
DOI: 10.1002/jnr.10089

Genes

GFAP

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 5 Sept 2026. Manifest publication-graph-cohort-2026-09-05.1.

Source receipt: pubmed, retrieved 5 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Upregulation of the HLH Id gene family in neural progenitors and glial cells of the rat spinal cord following contusion injury.PMID 11746449Read ↗

SOX2 expression is upregulated in adult spinal cord after contusion injury in both oligodendrocyte lineage and ependymal cellsSemantic proximity Read ↗

The Landscape of Gene Expression and Molecular Regulation Following Spinal Cord Hemisection in RatsSemantic proximity Read ↗

Interactions between ID and OLIG proteins mediate the inhibitory effects of BMP4 on oligodendroglial differentiation.Semantic proximity Read ↗

Combination strategies for repair, plasticity, and regeneration using regulation of gene expression during the chronic phase after spinal cord injurySemantic proximity Read ↗

The repair and autophagy mechanisms of hypoxia-regulated bFGF-modified primary embryonic neural stem cells in spinal cord injurySemantic proximity Read ↗

Time-Course Changes of Extracellular Matrix Encoding Genes Expression Level in the Spinal Cord Following Contusion Injury—A Data-Driven ApproachSemantic proximity Read ↗

Identification of genes induced in peripheral nerve after injury. Expression profiling and novel gene discovery.Semantic proximity Read ↗

Induction of ID2 Expression by Hypoxia-inducible Factor-1Semantic proximity Read ↗

Temporal and spatial pattern of DNA damage in neurons following spinal cord Injury in miceSemantic proximity Read ↗

NG2 colocalizes with axons and is expressed by a mixed cell population in spinal cord lesions.Semantic proximity Read ↗

Neuroprotective effect of endogenous pituitary adenylate cyclase-activating polypeptide on spinal cord injury.Semantic proximity Read ↗

Expression of Sam68 Associates with Neuronal Apoptosis and Reactive Astrocytes After Spinal Cord Injury.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.