Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

eNOS 894T allele and coronary blood flow at rest and during adenosine-induced hyperemia.

American journal of physiology. Heart and circulatory physiology - 1 Nov 2001

Naber C K, Baumgart D, Altmann C, Siffert W, Erbel R, Heusch G

Abstract excerpt

The 894T allele of a G894T polymorphism in the endothelial nitric oxide synthase (eNOS) gene is associated with decreased eNOS activity, cleavage of the protein, and endothelial dysfunction. The present study evaluated the association with coronary blood flow (CBF) at rest and during adenosine (ADO)-induced hyperemia. CBF was determined by Doppler flow wire and angiography in 97 left anterior descending arteries...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Adenosine
Share to Coronary Artery Disease

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Coronary Circulation

Share to Nitric Oxide Synthase

Share to Nitric Oxide Synthase Type III

Share to Ultrasonography

Share to Vasodilator Agents

Source

Identifiers

PMID: 11668050
DOI: 10.1152/ajpheart.2001.281.5.H1908

Genes

ADO

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

eNOS 894T allele and coronary blood flow at rest and during adenosine-induced hyperemia.PMID 11668050Read ↗

Effect of the 894G>T polymorphism of the endothelial nitric oxide synthase on vascular reactivity following maximal dynamic exercise.Semantic proximity Read ↗

Endothelial nitric oxide synthase Asp298Glu (894G/T) gene polymorphism as a possible risk factor for the coronary slow flow phenomenon among Iranians.Semantic proximity Read ↗

Interaction between endothelial nitric oxide synthase gene polymorphisms (-786T>C, 894G>T and intron 4 a/b) and cardiovascular risk factors in acute coronary syndromes.Semantic proximity Read ↗

Association of the 894G>T polymorphism of the endothelial constitutive nitric oxide synthase gene with unstable angina.Semantic proximity Read ↗

Coronary artery ectasia is associated with the c.894G>T (Glu298Asp) polymorphism of the endothelial nitric oxide synthase gene.Semantic proximity Read ↗

Influence of endothelial nitric oxide synthase gene polymorphisms (-786T>C, 4a4b, 894G>T) in Korean patients with coronary artery disease.Semantic proximity Read ↗

The 894G>T endothelial nitric oxide synthase genetic polymorphism affects hemodynamic responses to mental stress performed before and after exercise.Semantic proximity Read ↗

Plasma Nitric Oxide Concentrations and Nitric Oxide Synthase Gene Polymorphisms in Coronary Artery DiseaseSemantic proximity Read ↗

Genetic polymorphisms G894T on the eNOS gene is associated with endothelial function and vWF levels in premature myocardial infarction survivors.Semantic proximity Read ↗

The T-786C endothelial nitric oxide synthase genotype is a novel risk factor for coronary artery disease in Caucasian patients of the GENICA study.Semantic proximity Read ↗

Paraoxonase gene polymorphisms and coronary reactivity in young healthy men.Semantic proximity Read ↗

Influence of endothelial nitric oxide synthase gene polymorphisms (G894T, 4a4b, T-786C) and hyperhomocysteinemia on the predisposition to acute coronary syndromes.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.