Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Correlation of histologic subtypes and replication error phenotype with comparative genomic hybridization in gastric cancer.

Genes, chromosomes & cancer - 1 Jan 2001

Wu M S, Chang M C, Huang S P, Tseng C C, Sheu J C, Lin Y W, Shun C T, Lin M T, Lin J T

Abstract excerpt

To characterize phenotypic and genotypic changes in gastric cancer (GC), DNA copy number aberrations (CNAs) were assessed in 53 tumors using comparative genomic hybridization (CGH) and correlated with clinicopathologic characteristics and status of TP53 and replication error (RER). The number of CNAs per tumor was 6.8 (gain 5.3, loss 1.5), and the number of changes was significantly higher in tumors with advanced...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Chromosome Aberrations
Share to Disease Progression

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to DNA Replication

Share to Gene Dosage

Share to Genes, p53

Share to Nucleic Acid Hybridization

Share to Sequence Deletion

Share to Stomach Neoplasms

Source

Identifiers

PMID: 11107179
DOI: 10.1002/1098-2264(2000)9999:9999<::aid-gcc1062>3.0.co;2-r

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Correlation of histologic subtypes and replication error phenotype with comparative genomic hybridization in gastric cancer.PMID 11107179Read ↗

Clinical implications of chromosomal abnormalities in gastric adenocarcinomasSemantic proximity Read ↗

Novel regions of chromosomal amplification at 6p21, 5p13, and 12q14 in gastric cancer identified by array comparative genomic hybridizationSemantic proximity Read ↗

Genomic Alterations in Primary Gastric Adenocarcinomas Correlate with Clinicopathological Characteristics and SurvivalSemantic proximity Read ↗

Genome-wide analysis of DNA copy number alterations and gene expression in gastric cancer.Semantic proximity Read ↗

Integration of DNA Copy Number Alterations and Transcriptional Expression Analysis in Human Gastric CancerSemantic proximity Read ↗

Genomic profiling of gastric carcinoma in situ and adenomas by array-based comparative genomic hybridization.Semantic proximity Read ↗

Screening of DNA copy‐number aberrations in gastric cancer cell lines by array‐based comparative genomic hybridizationSemantic proximity Read ↗

Clinicopathological significance of altered loci of replication error and microsatellite instability-associated mutations in gastric cancer.Semantic proximity Read ↗

Exclusive Association of p53 Mutation with Super-High Methylation of Tumor Suppressor Genes in the p53 Pathway in a Unique Gastric Cancer Phenotype.Semantic proximity Read ↗

Gene Copy Number Change Events at Chromosome 20 and Their Association with Recurrence in Gastric Cancer PatientsSemantic proximity Read ↗

Frequent genomic copy number gain and overexpression of GATA-6 in pancreatic carcinomaSemantic proximity Read ↗

Oligonucleotide array comparative genomic hybridization refines the structure of 8p23.1, 17q12 and 20q13.2 amplifications in gastric carcinomas.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.