Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Incisional wound healing in transforming growth factor‐β1 null mice

1 Jun 2000

Abstract excerpt

Expression of endogenous transforming growth factor-beta1 is reduced in many animal models of impaired wound healing, and addition of exogenous transforming growth factor-beta has been shown to improve healing. To test the hypothesis that endogenous transforming growth factor-beta1 is essential for normal wound repair, we have studied wound healing in mice in which the transforming growth factor-beta1 gene has...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Tendon Structure and Treatment
Share to TGF-β signaling in diseases
Share to Wound Healing and Treatments

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Source

Identifiers

PMID: 10886809
DOI: 10.1046/j.1524-475x.2000.00179.x

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Incisional wound healing in transforming growth factor‐β1 null micePMID 10886809Read ↗

Transforming growth factor beta 1 null mutation in mice causes excessive inflammatory response and early death.Semantic proximity Read ↗

Absence of integrin-mediated TGFbeta1 activation in vivo recapitulates the phenotype of TGFbeta1-null mice.Semantic proximity Read ↗

Inflammation and TGF beta 1: lessons from the TGF beta 1 null mouse.Semantic proximity Read ↗

Wound healing and inflammation genes revealed by array analysis of 'macrophageless' PU.1 null miceSemantic proximity Read ↗

A model of scarless human fetal wound repair is deficient in transforming growth factor betaSemantic proximity Read ↗

Transforming growth factor-beta 1 knockout mice. A mutation in one cytokine gene causes a dramatic inflammatory disease.Semantic proximity Read ↗

IL-10 overexpression decreases inflammatory mediators and promotes regenerative healing in an adult model of scar formation.Semantic proximity Read ↗

Onset and progression of pathological lesions in transforming growth factor-beta 1-deficient mice.Semantic proximity Read ↗

Wound Healing in the α2β1 Integrin-Deficient Mouse: Altered Keratinocyte Biology and Dysregulated Matrix Metalloproteinase ExpressionSemantic proximity Read ↗

Aberrant Toll receptor expression and endotoxin hypersensitivity in mice lacking a functional TGF-beta 1 signaling pathway.Semantic proximity Read ↗

Delayed wound healing in the absence of intercellular adhesion molecule-1 or L-selectin expression.Semantic proximity Read ↗

Regulation of Wound Healing by Growth Factors and CytokinesSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.