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Article

BP180/type XVII collagen: its role in acquired and inherited disorders or the dermal-epidermal junction

9 Apr 1999

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Share to Autoimmune Bullous Skin Diseases
Share to Coagulation, Bradykinin, Polyphosphates, and Angioedema
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Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 10335914
DOI: 10.1007/s004030050392

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Related research

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Click a neighbor to travelStep 1 · 12 closest

BP180/type XVII collagen: its role in acquired and inherited disorders or the dermal-epidermal junctionPMID 10335914Read ↗

Collagen XVII.Semantic proximity Read ↗

BP180/Collagen XVII: A Molecular ViewSemantic proximity Read ↗

Type XV collagen exhibits a widespread distribution in human tissues but a distinct localization in basement membrane zonesSemantic proximity Read ↗

Collagen XVIII in tissue homeostasis and dysregulation — Lessons learned from model organisms and human patientsSemantic proximity Read ↗

Enigmatic insight into collagenSemantic proximity Read ↗

Deletion of the cytoplasmatic domain of BP180/collagen XVII causes a phenotype with predominant features of epidermolysis bullosa simplex.Semantic proximity Read ↗

Epidermolysis bullosa: Molecular pathology of connective tissue components in the cutaneous basement membrane zone.Semantic proximity Read ↗

[Dermis collagens: beyond their structural properties].Semantic proximity Read ↗

The impact of intrinsic ageing on the protein composition of the dermal-epidermal junctionSemantic proximity Read ↗

Heritable collagen disorders: from genotype to phenotype.Semantic proximity Read ↗

The collagenopathies: review of clinical phenotypes and molecular correlations.Semantic proximity Read ↗

From Structure to Phenotype: Impact of Collagen Alterations on Human Health.Semantic proximity Read ↗

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