Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

CD59-deficient blood cells and PIG-A gene abnormalities in Japanese patients with aplastic anaemia.

British journal of haematology - 1 Mar 1999

Azenishi Y, Ueda E, Machii T, Nishimura J, Hirota T, Shibano M, Nakao S, Kinoshita T, Mizoguchi H, Kitani T

Abstract excerpt

Patients with aplastic anaemia (AA) frequently develop paroxysmal nocturnal haemoglobinuria (PNH) as a late complication. We investigated the frequency of the development of PNH features including a glycosyl phosphatidylinositol (GPI) anchoring defect in 73 Japanese patients with AA. A deficient...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Japan
Share to Anemia, Aplastic
Share to CD59 Antigens

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Erythrocytes

Share to Glycosylphosphatidylinositols

Share to Granulocytes

Share to Hemoglobinuria, Paroxysmal

Share to Membrane Proteins

Source

Identifiers

PMID: 10086790
DOI: 10.1046/j.1365-2141.1999.01214.x

Genes

GPI

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 4 Sept 2026. Prepared record publication-10086790-2026-09-04.2. Manifest publication-graph-cohort-2026-09-04.2.

Source receipt: PubMed via Helena Literature Corpus, retrieved 4 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

CD59-deficient blood cells and PIG-A gene abnormalities in Japanese patients with aplastic anaemia.PMID 10086790Read ↗

High frequency of several PIG-A mutations in patients with aplastic anemia and myelodysplastic syndrome.Semantic proximity Read ↗

PIG-A gene mutations in four Taiwanese patients with paroxysmal nocturnal haemoglobinuria following aplastic anaemia.Semantic proximity Read ↗

Somatic mutations of the PIG-A gene found in Japanese patients with paroxysmal nocturnal hemoglobinuria.Semantic proximity Read ↗

Mutation analysis of the PIG-A gene in Korean patients with paroxysmal nocturnal haemoglobinuria.Semantic proximity Read ↗

The spectrum of PIG-A gene mutations in aplastic anemia/paroxysmal nocturnal hemoglobinuria (AA/PNH): a high incidence of multiple mutations and evidence of a mutational hot spot.Semantic proximity Read ↗

Phenotypes and phosphatidylinositol glycan-class A gene abnormalities during cell differentiation and maturation from precursor cells to mature granulocytes in patients with paroxysmal nocturnal hemoglobinuria.Semantic proximity Read ↗

The distribution of PIG-A gene abnormalities in paroxysmal nocturnal hemoglobinuria granulocytes and cultured erythroblasts.Semantic proximity Read ↗

Detection of GPI-anchored protein-deficient cells in patients with aplastic anaemia and evidence for clonal expansion during the clinical course.Semantic proximity Read ↗

Genotypic, immunophenotypic and clinical features of Thai patients with paroxysmal nocturnal haemoglobinuria.Semantic proximity Read ↗

Paroxysmal nocturnal hemoglobinuria: An acquired genetic disease.Semantic proximity Read ↗

PIG-A mutations in paroxysmal nocturnal hemoglobinuria and in normal hematopoiesis.Semantic proximity Read ↗

Characterization of genomic PIG-A gene: a gene for glycosylphosphatidylinositol-anchor biosynthesis and paroxysmal nocturnal hemoglobinuria.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.