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Lung tumors and non-malignant airways from patients with chronic lung inflammatory disease bear distinct genetic and epigenetic disruptions corresponding to metabolic processes involved in senescence and methylation

2014-01-01

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Literature Corpus work: 74191d12-7112-5397-a852-7d807d7f490d
DOI: 10.1186/2049-3002-2-s1-p82
PMCID: PMC4072978

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Lung tumors and non-malignant airways from patients with chronic lung inflammatory disease bear distinct genetic and epigenetic disruptions corresponding to metabolic processes involved in senescence and methylationDOI 10.1186/2049-3002-2-s1-p82Read

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