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Thermal and non-thermal stress conditions activate the <i>Plasmodium falciparum</i> AP2-HS-dependent heat-shock response

2025-11-24

Abstract excerpt

<h4>ABSTRACT</h4> To preserve proteome homeostasis and survive at higher-than-optimal temperatures, organisms have evolved the conserved heat shock (HS) response (HSR), characterised by increased expression of specific chaperone-encoding genes. In the human blood, malaria parasites are frequently exposed to elevated temperatures associated with host fever episodes. The protective HSR of Plasmodium falciparum , t...

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Literature Corpus work
4a6dc830-f566-5b7b-90e0-0f5e177fa636
DOI
10.1101/2025.11.24.690237
Open publication

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Thermal and non-thermal stress conditions activate the <i>Plasmodium falciparum</i> AP2-HS-dependent heat-shock responseDOI 10.1101/2025.11.24.690237
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