The [KIL-d] cytoplasmic genetic element of yeast results in epigenetic regulation of viral M double-stranded RNA gene expression.

doi:10.1093/genetics/150.1.21

Back to search

Article

The [KIL-d] cytoplasmic genetic element of yeast results in epigenetic regulation of viral M double-stranded RNA gene expression.

Genetics - 1 Sept 1998

Tallóczy Z, Menon S, Neigeborn L, Leibowitz M J

Abstract excerpt

[KIL-d] is a cytoplasmically inherited genetic trait that causes killer virus-infected cells of Saccharomyces cerevisiae to express the normal killer phenotypes in a/alpha cells, but to show variegated defective killer phenotypes in a or alpha type cells. Mating of [KIL-d] haploids results in "he...

Topics

Base Sequence
DNA Primers
Gene Expression Regulation, Fungal
Haploidy
Heterozygote
Mating Factor
Mutation
Peptides
Phenotype
RNA Viruses
RNA, Double-Stranded
RNA, Fungal
Saccharomyces cerevisiae

Community interpretation

Discuss the evidence

Ask a question, add context or challenge an interpretation. Community activity is separate from scientific evidence strength.

discussions

replies

Citations encouraged

Claims remain attributable.

No discussion yet

Be the first to add a careful, cited interpretation.