Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Human papillomavirus E6 and E7 oncoproteins alter cell cycle progression but not radiosensitivity of carcinoma cells treated with low-dose-rate radiation

1 Jan 1997

Abstract excerpt

No abstract is available from the source.

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cancer-related Molecular Pathways
Share to Effects of Radiation Exposure
Share to Endometrial and Cervical Cancer Treatments

Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 9054890
DOI: 10.1016/s0360-3016(96)00448-8

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 8 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Human papillomavirus E6 and E7 oncoproteins alter cell cycle progression but not radiosensitivity of carcinoma cells treated with low-dose-rate radiationPMID 9054890Read ↗

Human papillomavirus E6/E7 oncoproteins promote radiotherapy-mediated tumor suppression by globally hijacking host DNA damage repairSemantic proximity Read ↗

Molecular mechanisms underlying human papillomavirus E6 and E7 oncoprotein-induced cell transformationSemantic proximity Read ↗

Radiosensitization of Oropharyngeal Squamous Cell Carcinoma Cells by Human Papillomavirus 16 Oncoprotein E6∗ISemantic proximity Read ↗

Human papillomavirus type 16 E7 oncoprotein causes a delay in repair of DNA damageSemantic proximity Read ↗

Poor prognosis associated with human papillomavirus α7 genotypes in cervical carcinoma cannot be explained by intrinsic radiosensitivity.Semantic proximity Read ↗

Human papillomavirus type 16 <i>E6</i> and <i>E7</i> oncogenes abrogate radiation-induced DNA damage responses <i>in vivo</i> through p53-dependent and p53-independent pathwaysSemantic proximity Read ↗

Radiation-enhanced expression of E6/E7 transforming oncogenes of human papillomavirus-16 in human cervical carcinomaSemantic proximity Read ↗

Human papillomavirus E1 and E2 mediated DNA replication is not arrested by DNA damage signallingSemantic proximity Read ↗

p53 mutations and presence of HPV DNA do not correlate with radiosensitivity of gynecological cancer cell lines.Semantic proximity Read ↗

Cellular responses of HPV-positive cancer cells to genotoxic anti-cancer agents: Repression ofE6/E7-oncogene expression and induction of apoptosisSemantic proximity Read ↗

Growth arrest by induction of p53 in DNA damaged keratinocytes is bypassed by human papillomavirus 16 E7.Semantic proximity Read ↗

Human papillomavirus 16 E6 increases the radiosensitivity of p53-mutated cervical cancer cells, associated with up-regulation of aurora A.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.