Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Measles virus replication in neural cells.

Trends in microbiology - 1 Sept 1995

Johnston I C, Dunster L M, Schneider-Schaulies J, Schneider-Schaulies S

Abstract excerpt

Measles virus gene expression is attenuated in neural cells by mechanisms that affect both viral transcription and translation. Host enzymes that hypermutate viral genes, and those induced by cytokines, may act cooperatively to slow viral replication and to favor persistent measles virus infections in the human central nervous system.

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Brain
Share to Cytokines
Share to Gene Expression Regulation, Viral

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Genes, Viral

Share to Mutation

Share to Neurons

Share to Transcription, Genetic

Share to Virus Replication

Source

Identifiers

PMID: 8520890
DOI: 10.1016/s0966-842x(00)88976-3

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Measles virus replication in neural cells.PMID 8520890Read ↗

Molecular and cellular aspects of measles virus persistence in the CNS.Semantic proximity Read ↗

Impaired Cholesterol Biosynthesis in a Neuronal Cell Line Persistently Infected with Measles VirusSemantic proximity Read ↗

Suppression of viral RNA polymerase activity is necessary for persistent infection during the transformation of measles virus into SSPE virusSemantic proximity Read ↗

Evidence That the Hypermutated M Protein of a Subacute Sclerosing Panencephalitis Measles Virus Actively Contributes to the Chronic Progressive CNS DiseaseSemantic proximity Read ↗

Clonal Expansion of Hypermutated Measles Virus in a SSPE BrainSemantic proximity Read ↗

Generation and properties of measles virus mutations typically associated with subacute sclerosing panencephalitis.Semantic proximity Read ↗

New Insights into Measles Virus Brain InfectionsSemantic proximity Read ↗

Host factors and measles virus replicationSemantic proximity Read ↗

Measles Virus Neurovirulence and Host ImmunitySemantic proximity Read ↗

Defective Measles Virus in Human Subacute Sclerosing Panencephalitis BrainSemantic proximity Read ↗

Modeling subacute sclerosing panencephalitis in a transgenic mouse system: uncoding pathogenesis of disease and illuminating components of immune control.Semantic proximity Read ↗

Full-length sequence analysis of subacute sclerosing panencephalitis (SSPE) virus, a mutant of measles virus, isolated from brain tissues of a patient shortly after onset of SSPE.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.