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Article

Calcium/calmodulin-dependent protein kinase II and potassium channel subunit eag similarly affect plasticity in Drosophila.

Proceedings of the National Academy of Sciences of the United States of America - 11 Oct 1994

Griffith L C, Wang J, Zhong Y, Wu C F, Greenspan R J

Abstract excerpt

Similar defects in both synaptic transmission and associative learning are produced in Drosophila melanogaster by inhibition of calcium/calmodulin-dependent protein kinase II and mutations in the potassium channel subunit gene eag. These behavioral and synaptic defects are not simply additive in animals carrying both an eag mutation and a transgene for a protein kinase inhibitor, raising the possibility that the...

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PMID: 7937834
DOI: 10.1073/pnas.91.21.10044
PMCID: PMC44954

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