Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Maternal influence on susceptibility of offspring to Brugia malayi infection in a murine model of filariasis.

Acta tropica - 1 Dec 1994

Rajan T V, Bailis J M, Yates J A, Shultz L D, Greiner D L, Nelson F K

Abstract excerpt

We have used the severe combined immunodeficient C.B-17-scid/scid mouse to investigate the influences of maternal immune status and parasite burden on the susceptibility (or resistance) of offspring to infection with the human filarial parasite, Brugia malayi. C.B-17-scid/scid mice are permissive for infection while immunocompetent C.B-17(-)+/+ mice are uniformly resistant. Reciprocal matings of C.B-17-scid/scid...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Alleles
Share to Animals, Newborn
Share to Genetic Predisposition to Disease

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to Mice, SCID

Source

Identifiers

PMID: 7709867
DOI: 10.1016/0001-706x(94)90022-1

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Maternal influence on susceptibility of offspring to Brugia malayi infection in a murine model of filariasis.PMID 7709867Read ↗

T cells facilitate Brugia malayi development in TCRalpha(null) mice.Semantic proximity Read ↗

The immunodeficient scid mouse as a model for human lymphatic filariasis.Semantic proximity Read ↗

Genes from Chagas susceptibility loci that are differentially expressed in T. cruzi-resistant mice are candidates accounting for impaired immunity.Semantic proximity Read ↗

Maternal Helminth Infection Causes Dysfunctional B Cell Development in Male OffspringSemantic proximity Read ↗

Mosquito Infection Responses to Developing Filarial WormsSemantic proximity Read ↗

Pregnancy impairs resistance of C57BL/6 mice to Leishmania major infection and causes decreased antigen-specific IFN-gamma response and increased production of T helper 2 cytokines.Semantic proximity Read ↗

Sex-dependent genetic effects on immune responses to a parasitic nematode.Semantic proximity Read ↗

Innate Resistance toBabesiaInfection Is Influenced by Genetic Background and GenderSemantic proximity Read ↗

Evidence for genomic imprinting of the major QTL controlling susceptibility to trypanosomiasis in mice.Semantic proximity Read ↗

Genetic susceptibility to vaginal candidiasis.Semantic proximity Read ↗

Chromosomal mapping of two loci affecting filarial worm susceptibility in Aedes aegypti.Semantic proximity Read ↗

Do mice genetically selected for resistance to oral tolerance provide selective advantage for Schistosoma mansoni infection?Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.