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Article

Host-induced climate change: Carbon dioxide tolerance as a Cryptococcus neoformans virulence trait

8 Aug 2025

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 40779504
DOI: 10.1371/journal.ppat.1013351

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Host-induced climate change: Carbon dioxide tolerance as a Cryptococcus neoformans virulence traitPMID 40779504Read ↗

Host Carbon Dioxide Concentration Is an Independent Stress for Cryptococcus neoformans That Affects Virulence and Antifungal SusceptibilitySemantic proximity Read ↗

Host carbon dioxide concentration is an independent stress for <i>Cryptococcus neoformans</i> that affects virulence and antifungal susceptibilitySemantic proximity Read ↗

Discovery of CO2 tolerance genes associated with virulence in the fungal pathogen Cryptococcus neoformansSemantic proximity Read ↗

Microevolution of Cryptococcus neoformans in high CO2 converges on mutations isolated from patients with relapsed cryptococcosis.Semantic proximity Read ↗

<i>Cryptococcus neoformans</i> adapts to host CO <sub>2</sub> concentrations through the coordinated remodeling of central carbon metabolism, oxidative stress resistance, and membrane homeostasisSemantic proximity Read ↗

Cryptococcus neoformans adapts to the host environment through TOR-mediated remodeling of phospholipid asymmetrySemantic proximity Read ↗

Adaptation of Cryptococcus neoformans to Mammalian Hosts: Integrated Regulation of Metabolism and VirulenceSemantic proximity Read ↗

Carbon dioxide is a powerful inducer of monokaryotic hyphae and spore development in Cryptococcus gattii and carbonic anhydrase activity is dispensable in this dimorphic transition.Semantic proximity Read ↗

Distinct routes to thermotolerance in the fungal pathogen Cryptococcus neoformans.Semantic proximity Read ↗

Cryptococcus neoformans, a fungus under stressSemantic proximity Read ↗

Amoeba predation of <i>Cryptococcus neoformans</i> results in pleiotropic changes to traits associated with virulenceSemantic proximity Read ↗

Pan-drug resistance and hypervirulence in a human fungal pathogen are enabled by mutagenesis induced by mammalian body temperatureSemantic proximity Read ↗

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