Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Aberrant T-cell phenotypes in a cohort of patients with post-treatment Lyme disease

9 Jul 2025

Abstract excerpt

infection with significant patient morbidity. Characterized by fatigue, generalized myalgias, and cognitive impairment, PTLD symptomatology closely resembles long COVID and other post-acute infection syndromes. While prior studies suggest immune dysregulation as a factor in PTLD pathogenesis, the mechanisms underlying its heterogeneous presentation and severity remain unclear. To associate symptom burden with...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Toxin Mechanisms and Immunotoxins
Share to Vector-borne infectious diseases
Share to Viral Infections and Vectors

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 40703523
DOI: 10.3389/fimmu.2025.1607619

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 6 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Aberrant T-cell phenotypes in a cohort of patients with post-treatment Lyme diseasePMID 40703523Read ↗

Persistent T cell phenotypic alterations and early innate immune dysregulation as potential biomarkers of Long COVID.Semantic proximity Read ↗

Cytokine Hub Classification of PASC, ME-CFS and other PASC-like ConditionsSemantic proximity Read ↗

Severe fatigue as symptom of long COVID is characterized by increased expression of inflammatory genes in monocytes, increased serum pro-inflammatory cytokines, and increased CD8+ T-lymphocytes: A putative dysregulation of the immune-brain axis, the coagulation process, and auto-inflammation to explain the diversity of long COVID symptomsSemantic proximity Read ↗

Blood T cell phenotypes correlate with fatigue severity in post-acute sequelae of COVID-19.Semantic proximity Read ↗

Abnormal T-Cell Activation And Cytotoxic T-Cell Frequency Discriminates Symptom Severity In Myalgic Encephalomyelitis/Chronic Fatigue SyndromeSemantic proximity Read ↗

RNA-Seq Analysis of Gene Expression, Viral Pathogen, and B-Cell/T-Cell Receptor Signatures in Complex Chronic Disease.Semantic proximity Read ↗

Gene set predictor for post-treatment Lyme diseaseSemantic proximity Read ↗

Longitudinal immune profiling of Long COVID phenotypes reveals a shared persistent IL-17F-centered immune signatureSemantic proximity Read ↗

HERV activation segregates ME/CFS from fibromyalgia while defining a novel nosologic entitySemantic proximity Read ↗

Changes in the transcriptome of circulating immune cells of a New Zealand cohort with myalgic encephalomyelitis/chronic fatigue syndromeSemantic proximity Read ↗

The Female Predominant Persistent Immune Dysregulation of the Post COVID Syndrome: A Cohort StudySemantic proximity Read ↗

Single-cell transcriptomics of blood identified IFIT1 neutrophil subcluster expansion in NTM-PD patientsSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.