Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Omics-based analysis of mitochondrial dysfunction and BBB integrity in post-COVID-19 sequelae

28 Dec 2024

Abstract excerpt

The SARS-CoV-2 virus that resulted in the COVID-19 pandemic has been implicated in a range of neurological issues, such as encephalopathy, stroke, and cognitive decline. Although the precise mechanism causing these issues is unknown, mounting evidence shows that blood-brain barrier (BBB) disruption is probable2 a major factor. The integrity of the blood-brain barrier (BBB), a highly selective barrier that divides...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Infectious Encephalopathies and Encephalitis
Share to Long-Term Effects of COVID-19
Share to Neuroinflammation and Neurodegeneration Mechanisms

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 39730725
DOI: 10.1038/s41598-024-82180-6

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 2 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Omics-based analysis of mitochondrial dysfunction and BBB integrity in post-COVID-19 sequelaePMID 39730725Read ↗

SARS-CoV-2 infection of human brain microvascular endothelial cells leads to inflammatory activation through NF-κB non-canonical pathway and mitochondrial remodelingSemantic proximity Read ↗

Human Brain Microvascular Endothelial Cells Exposure to SARS-CoV-2 Leads to Inflammatory Activation through NF-κB Non-Canonical Pathway and Mitochondrial RemodelingSemantic proximity Read ↗

SARS-CoV-2 infection of human brain microvascular endothelial cells leads to inflammatory activation through NF-κB non-canonical pathway and mitochondrial remodelingSemantic proximity Read ↗

Detection of SARS-CoV-2 genome and whole transcriptome sequencing in frontal cortex of COVID-19 patientsSemantic proximity Read ↗

SARS-CoV-2 infects brain astrocytes of COVID-19 patients and impairs neuronal viabilitySemantic proximity Read ↗

Bioinformatics and system biology approaches to identify pathophysiological impact of COVID-19 to the progression and severity of neurological diseasesSemantic proximity Read ↗

Single-Cell Transcriptomics Reveals Evidence of Endothelial Dysfunction in the Brains of COVID-19 Patients with Implications for Glioblastoma ProgressionSemantic proximity Read ↗

SARS-CoV-2 productively infects human brain microvascular endothelial cellsSemantic proximity Read ↗

Brain-wide alterations revealed by spatial transcriptomic and proteomic analyses in COVID-19 infectionSemantic proximity Read ↗

Pathophysiological involvement of host mitochondria in SARS-CoV-2 infection that causes COVID-19: a comprehensive evidential insightSemantic proximity Read ↗

Investigating Molecular Aspects of SARS-CoV-2 Neurological Manifestations, a Systems Biology ApproachSemantic proximity Read ↗

Decoding SARS-CoV-2 hijacking of host mitochondria in COVID-19 pathogenesisSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.