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Maternal 129S1/SvImJ background attenuates the placental phenotypes induced by chronic paternal alcohol exposure.

Reproductive toxicology (Elmsford, N.Y.) - 1 Jun 2024

Bhadsavle Sanat S, Scaturro Katherine Z, Golding Michael C

Abstract excerpt

Paternal alcohol use is emerging as a plausible driver of alcohol-related growth and patterning defects. Studies from our lab using an inbred C57Bl/6 J mouse model suggest that these paternally-inherited phenotypes result from paternally programmed deficits in the formation and function of the placenta. The 129S1/SvImJ genetic background is typically more susceptible to fetoplacental growth defects due to...

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PMID: 38735594
DOI: 10.1016/j.reprotox.2024.108605

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