Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Secondary messenger signalling influences Pseudomonas aeruginosa adaptation to sinus and lung environments.

The ISME journal - 8 Jan 2024

Ruhluel Dilem, Fisher Lewis, Barton Thomas E, Leighton Hollie, Kumar Sumit, Amores Morillo Paula, O'Brien Siobhan, Fothergill Joanne L, Neill Daniel R

Abstract excerpt

Pseudomonas aeruginosa is a cause of chronic respiratory tract infections in people with cystic fibrosis (CF), non-CF bronchiectasis, and chronic obstructive pulmonary disease. Prolonged infection allows the accumulation of mutations and horizontal gene transfer, increasing the likelihood of adaptive phenotypic traits. Adaptation is proposed to arise first in bacterial populations colonizing upper airway...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Biofilms
Share to Cyclic GMP

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Cystic Fibrosis

Share to Fimbriae, Bacterial

Share to Pseudomonas aeruginosa

Share to Pseudomonas Infections

Share to Second Messenger Systems

Source

Open in PubMed Open full text

Identifiers

PMID: 38647527
DOI: 10.1093/ismejo/wrae065
PMCID: PMC11102083

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Secondary messenger signalling influences Pseudomonas aeruginosa adaptation to sinus and lung environments.PMID 38647527Read ↗

Pseudomonas aeruginosa Diversification during Infection Development in Cystic Fibrosis Lungs—A ReviewSemantic proximity Read ↗

Rapid diversification of <i>Pseudomonas aeruginosa</i> in cystic fibrosis lung-like conditionsSemantic proximity Read ↗

The nutritional environment is sufficient to select coexisting biofilm and quorum-sensing mutants of <i>Pseudomonas aeruginosa</i>Semantic proximity Read ↗

Rapid diversification of Pseudomonas aeruginosa in cystic fibrosis lung-like conditionsSemantic proximity Read ↗

Pseudomonas aeruginosa infection and persistence mechanisms in cystic fibrosis patientsSemantic proximity Read ↗

Infection dynamics and virulence potential of clinical <i>Pseudomonas aeruginosa</i> isolates in human airway epithelial modelsSemantic proximity Read ↗

Differential adaptation of microbial pathogens to airways of patients with cystic fibrosis and chronic obstructive pulmonary diseaseSemantic proximity Read ↗

Biofilm Formation of Pseudomonas aeruginosa in Cystic Fibrosis: Mechanisms of Persistence, Adaptation, and PathogenesisSemantic proximity Read ↗

Development of liquid culture media mimicking the conditions of sinuses and lungs in cystic fibrosis and healthSemantic proximity Read ↗

Development of liquid culture media mimicking the conditions of sinuses and lungs in cystic fibrosis and healthSemantic proximity Read ↗

Evolution and adaptation of <i>Pseudomonas aeruginosa</i> in the paranasal sinuses of people with cystic fibrosisSemantic proximity Read ↗

Intrinsic and environmental mutagenesis drive diversification and persistence of Pseudomonas aeruginosa in chronic lung infections.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.