Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

The LINC01176-miR-218-5p-IL-36G Network is Responsible for the Pathogenesis of Psoriasis by Promoting Inflammation

1 Jan 2024

Abstract excerpt

Purpose: Psoriasis is an incurable chronic inflammatory skin disease. The exact function and regulatory mechanism of non-coding RNA upregulation in psoriasis remains to be elucidated. The aim of this study was to analyse the role of the lncRNA-miRNA-mRNA network of psoriasis and LINC01176 in the pathogenesis of psoriasis. Patients and Methods: We performed miRNA, lncRNA, and mRNA sequencing analysis in...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cancer-related molecular mechanisms research
Share to Cytokine Signaling Pathways and Interactions
Share to Psoriasis: Treatment and Pathogenesis

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Source

Identifiers

PMID: 38193028
DOI: 10.2147/ccid.s444265

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 8 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

The LINC01176-miR-218-5p-IL-36G Network is Responsible for the Pathogenesis of Psoriasis by Promoting InflammationPMID 38193028Read ↗

miRNAs Flowing Up and Down: The Concerto of PsoriasisSemantic proximity Read ↗

Downregulated miR‐187 contributes to the keratinocytes hyperproliferation in psoriasisSemantic proximity Read ↗

The role of microRNA in psoriasis: A reviewSemantic proximity Read ↗

The Benefit of the Optimized Formula of Yinxieling in Psoriasis Vulgaris via Regulation on Autophagy Based on microRNA Expression Profile and Network Pharmacology AnalysisSemantic proximity Read ↗

Transcriptional landscape of psoriasis identifies the involvement of IL36 and IL36RNSemantic proximity Read ↗

miR-146b Probably Assists miRNA-146a in the Suppression of Keratinocyte Proliferation and Inflammatory Responses in PsoriasisSemantic proximity Read ↗

Identification of key genes and Construction of miRNA-mRNA regulatory network in Psoriasis based on Bioinformatics AnalysisSemantic proximity Read ↗

miR-383 reduces keratinocyte proliferation and induces the apoptosis in psoriasis via disruption of LCN2-dependent JAK/STAT pathway activationSemantic proximity Read ↗

An Exploration of the Role of MicroRNAs in PsoriasisSemantic proximity Read ↗

Construction of a lncRNA‑miRNA‑mRNA network to determine the regulatory roles of lncRNAs in psoriasisSemantic proximity Read ↗

Integrated analysis of immune-related long noncoding RNAs as diagnostic biomarkers in psoriasisSemantic proximity Read ↗

LncRNA lnc-SPRR2G-2 contributes to keratinocyte hyperproliferation and inflammation in psoriasis by activating the STAT3 pathway and downregulating KHSRPSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.