Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

TLRs: Innate Immune Sentries against SARS-CoV-2 Infection

29 Apr 2023

Abstract excerpt

Coronavirus disease 2019 (COVID-19), caused by severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), has been responsible for a devastating pandemic since March 2020. Toll-like receptors (TLRs), crucial components in the initiation of innate immune responses to different pathogens, trigger the downstream production of pro-inflammatory cytokines, interferons, and other mediators. It has been demonstrated...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to COVID-19 Clinical Research Studies
Share to Respiratory Support and Mechanisms
Share to Sepsis Diagnosis and Treatment

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 37175768
DOI: 10.3390/ijms24098065

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 8 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

TLRs: Innate Immune Sentries against SARS-CoV-2 InfectionPMID 37175768Read ↗

Toll-like receptor (TLRs) agonists and antagonists for COVID-19 treatmentsSemantic proximity Read ↗

Interplay of TLR4 and SARS-CoV-2: Unveiling the Complex Mechanisms of Inflammation and Severity in COVID-19 InfectionsSemantic proximity Read ↗

SARS-CoV-2 activates the TLR4/MyD88 pathway in human macrophages: A possible correlation with strong pro-inflammatory responses in severe COVID-19Semantic proximity Read ↗

SARS-CoV-2-associated ssRNAs activate inflammation and immunity via TLR7/8Semantic proximity Read ↗

Overly Exuberant Innate Immune Response to SARS-CoV-2 InfectionSemantic proximity Read ↗

Pattern Recognition Receptors Expression and Activation in COVID-19 and Long COVID: From SARS-CoV-2 Escape Mechanisms to Emerging ImmunotherapiesSemantic proximity Read ↗

IMMUNE RESPONSE TO SARS CoV2 INFECTION BY TLR3, TLR4 AND TLR7 GENE EXPRESSIONSemantic proximity Read ↗

IMMUNE RESPONSE TO SARS CoV2 INFECTION BY TLR3, TLR4 AND TLR7 GENE EXPRESSION.Semantic proximity Read ↗

Adaptive Immunity to Viruses: What Did We Learn from SARS-CoV-2 Infection?Semantic proximity Read ↗

Mechanistic insight into the protective and pathogenic immune-responses against SARS-CoV-2.Semantic proximity Read ↗

Nucleic Acid-Sensing Pathways During SARS-CoV-2 Infection: Expectations versus RealitySemantic proximity Read ↗

Dysregulation of Immune Response in Patients with COVID-19 in Wuhan, ChinaSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.