Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

RNA sequencing and integrative analysis reveal pathways and hub genes associated with TGFβ1 stimulation on prostatic stromal cells

12 Aug 2022

Abstract excerpt

Objective: Benign prostatic hyperplasia (BPH) is the most common urological disease in elderly men. The transforming growth factor beta 1 (TGFβ1) plays an important role in the proliferation and differentiation of BPH stroma. However, it is not clear yet which important pathways and key genes are the downstream of TGFβ1 acting on prostatic stromal cells. Methods: GSE132714 is currently the newer, available, and...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Phytoestrogen effects and research
Share to Prostate Cancer Diagnosis and Treatment
Share to Prostate Cancer Treatment and Research

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 36035183
DOI: 10.3389/fgene.2022.919103

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 2 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

RNA sequencing and integrative analysis reveal pathways and hub genes associated with TGFβ1 stimulation on prostatic stromal cellsPMID 36035183Read ↗

Long Noncoding RNA SNHG16 Functions as Tumor Activator by Sponging hsa-miR-373-3p to Regulate TGFBR2/SMAD Pathway in Prostate CancerSemantic proximity Read ↗

Genomic analysis of benign prostatic hyperplasia implicates cellular relandscaping in disease pathogenesisSemantic proximity Read ↗

A transition zone enriched <i>WIF1</i> ⁺ basal cell subtype is associated with benign prostatic hyperplasiaSemantic proximity Read ↗

Identification of Potential Key Genes in Prostate Cancer with Gene Expression, Pivotal Pathways and Regulatory Networks Analysis Using Integrated Bioinformatics MethodsSemantic proximity Read ↗

Integrative exploration of the mutual gene signatures and immune microenvironment between benign prostate hyperplasia and castration-resistant prostate cancerSemantic proximity Read ↗

SRD5A2 in the development of benign prostatic hyperplasia mechanism and immune infiltration analysisSemantic proximity Read ↗

Single cell transcriptional profiling of benign prostatic hyperplasia reveals a progenitor-like luminal epithelial cell state within an inflammatory microenvironmentSemantic proximity Read ↗

Elucidation of Inflammatory-Fibrotic Microenvironment in Human Urethral Stricture DiseaseSemantic proximity Read ↗

TGFBR3 loss and consequences in prostate cancerSemantic proximity Read ↗

TGF-β and microRNA Interplay in Genitourinary CancersSemantic proximity Read ↗

Identification of hub genes and their clinical value for predicting the development of prostate cancer from benign prostate hyperplasia by bioinformatic analysisSemantic proximity Read ↗

TIAM1 signaling drives prostatic budding and branching phenotypes and is a potential therapeutic target for BPHSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.