Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

SCFD1 expression quantitative trait loci in amyotrophic lateral sclerosis are differentially expressed

1 Oct 2021

Abstract excerpt

Abstract Evidence indicates that common variants found in genome-wide association studies increase risk of disease through gene regulation via expression Quantitative Trait Loci. Using multiple genome-wide methods, we examined if Single Nucleotide Polymorphisms increase risk of Amyotrophic Lateral Sclerosis through expression Quantitative Trait Loci, and whether expression Quantitative Trait Loci expression is...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Amyotrophic Lateral Sclerosis Research
Share to Epigenetics and DNA Methylation
Share to Neurogenetic and Muscular Disorders Research

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 34708205
DOI: 10.1093/braincomms/fcab236

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 4 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

SCFD1 expression quantitative trait loci in amyotrophic lateral sclerosis are differentially expressedPMID 34708205Read ↗

Common and rare variant association analyses in amyotrophic lateral sclerosis identify 15 risk loci with distinct genetic architectures and neuron-specific biology.Semantic proximity Read ↗

Genetic analysis of amyotrophic lateral sclerosis identifies contributing pathways and cell typesSemantic proximity Read ↗

Genetic analysis of amyotrophic lateral sclerosis identifies contributing pathways and cell types.Semantic proximity Read ↗

Common and rare variant association analyses in Amyotrophic Lateral Sclerosis identify 15 risk loci with distinct genetic architectures and neuron-specific biologySemantic proximity Read ↗

Common and rare variant association analyses in Amyotrophic Lateral Sclerosis identify 15 risk loci with distinct genetic architectures and neuron-specific biologySemantic proximity Read ↗

Genome-wide Identification of the Genetic Basis of Amyotrophic Lateral SclerosisSemantic proximity Read ↗

Genome-wide identification of the genetic basis of amyotrophic lateral sclerosisSemantic proximity Read ↗

Causal Inference of Genetic Variants and Genes in Amyotrophic Lateral SclerosisSemantic proximity Read ↗

Genetics of Amyotrophic Lateral SclerosisSemantic proximity Read ↗

Partitioning the genetic architecture of amyotrophic lateral sclerosisSemantic proximity Read ↗

Identification of potential drug targets for amyotrophic lateral sclerosis by Mendelian randomization analysis based on brain and plasma proteomics.Semantic proximity Read ↗

Gene co-expression network analysis in human spinal cord highlights mechanisms underlying amyotrophic lateral sclerosis susceptibilitySemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.