Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Determinants of expression of SARS‐CoV‐2 entry‐related genes in upper and lower airways

26 Oct 2021

Abstract excerpt

To the Editor, The coronavirus disease 2019 (COVID-19) pandemic is caused by severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2). To better understand COVID-19 the genetic and environmental factors on susceptibility and severity, detailed knowledge of regulation of genes required for viral entry into respiratory epithelial cells is needed. We assessed the gene expression of SARS-CoV-2 receptors and...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to COVID-19 Clinical Research Studies
Share to Respiratory viral infections research
Share to SARS-CoV-2 and COVID-19 Research

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 34698405
DOI: 10.1111/all.15152

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 5 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Determinants of expression of SARS‐CoV‐2 entry‐related genes in upper and lower airwaysPMID 34698405Read ↗

Determinants of SARS-CoV-2 receptor gene expression in upper and lower airwaysSemantic proximity Read ↗

Smoking modulates different secretory subpopulations expressing SARS-CoV-2 entry genes in the nasal and bronchial airwaysSemantic proximity Read ↗

Smoking Modulates Different Secretory Subpopulations Expressing SARS-CoV-2 Entry Genes in the Nasal and Bronchial AirwaysSemantic proximity Read ↗

Expression of ACE2, TMPRSS2 and CTSL in human airway epithelial cells under physiological and pathological conditions: Implications for SARS-CoV-2 infectionSemantic proximity Read ↗

Association of Cigarette Smoking, COPD, and Lung Cancer With Expression of SARS-CoV-2 Entry Genes in Human Airway Epithelial CellsSemantic proximity Read ↗

A Hint on the COVID-19 Risk: Population Disparities in Gene Expression of Three Receptors of SARS-CoVSemantic proximity Read ↗

ACE-2 Expression in the Small Airway Epithelia of Smokers and COPD Patients: Implications for COVID-19Semantic proximity Read ↗

Integrated analyses of single-cell atlases reveal age, gender, and smoking status associations with cell type-specific expression of mediators of SARS-CoV-2 viral entry and highlights inflammatory programs in putative target cellsSemantic proximity Read ↗

SARS-CoV-2 receptor ACE2 and TMPRSS2 are primarily expressed in bronchial transient secretory cellsSemantic proximity Read ↗

Genetic and non-genetic factors affecting the expression of COVID-19-relevant genes in the large airway epithelium.Semantic proximity Read ↗

Tobacco Smoking Increases the Lung Gene Expression of ACE2, the Receptor of SARS-CoV-2Semantic proximity Read ↗

Comparative Study of Epithelial Entry Gene Expression of SARS-Cov-2 and Other Human Viral Species in Asthma: Differences by Sex, Airway Location Aand Disease EndotypeSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.