Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

HIF1A polymorphisms do not modify the risk of epilepsy nor cerebral palsy after neonatal hypoxic-ischemic encephalopathy.

Brain research - 15 Apr 2021

Kukec Eva, Goričar Katja, Dolžan Vita, Rener-Primec Zvonka

Abstract excerpt

PURPOSE: Hypoxic-ischemic encephalopathy (HIE) remains the major cause of cerebral palsy and epilepsy in developed countries. Hypoxia-inducible factor 1 alpha (HIF-1α) is the key mediator of oxygen homoeostasis. The aim of this study was to investigate whether hypoxia-inducible factor 1 subunit alpha (HIF1A) functional polymorphisms are associated with the risk of epilepsy, drug-resistant epilepsy, and cerebral...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cerebral Palsy
Share to Drug Resistance
Share to Epilepsy

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Genetic Predisposition to Disease

Share to Hypoxia-Inducible Factor 1, alpha Subunit

Share to Hypoxia-Ischemia, Brain

Share to Infant, Newborn

Share to Polymorphism, Single Nucleotide

Source

Identifiers

PMID: 33515534
DOI: 10.1016/j.brainres.2021.147281

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 6 Sept 2026. Manifest publication-graph-cohort-2026-09-06.1.

Source receipt: pubmed, retrieved 6 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

HIF1A polymorphisms do not modify the risk of epilepsy nor cerebral palsy after neonatal hypoxic-ischemic encephalopathy.PMID 33515534Read ↗

Exploring the genetic landscape of neurotransmitter alterations in hypoxic-ischemic encephalopathy: A personalized medicine perspectiveSemantic proximity Read ↗

Antioxidant polymorphisms do not influence the risk of epilepsy or its drug resistance after neonatal hypoxic-ischemic brain injury.Semantic proximity Read ↗

Genetic Diagnosis in Neonatal Encephalopathy With Hypoxic Brain Damage Using Targeted Gene Panel SequencingSemantic proximity Read ↗

Genetic and epigenetic contributors and mimickers of phenotypic hypoxic-ischemic encephalopathy (HIE).Semantic proximity Read ↗

Clinical Implications of Epigenetic Dysregulation in Perinatal Hypoxic-Ischemic Brain DamageSemantic proximity Read ↗

Identifying Genetic Susceptibility in Neonates With Hypoxic-Ischemic Encephalopathy: A Retrospective Case Series.Semantic proximity Read ↗

Hypoxia-Inducible Pathway Polymorphisms and Their Role in the Complications of Prematurity.Semantic proximity Read ↗

The association between antioxidant enzyme polymorphisms and cerebral palsy after perinatal hypoxic-ischaemic encephalopathy.Semantic proximity Read ↗

Neonatal Hypoxic-Ischemic Encephalopathy Diagnosis and Treatment: A National Survey in ChinaSemantic proximity Read ↗

Single-nucleotide polymorphism screening and RNA sequencing of key messenger RNAs associated with neonatal hypoxic-ischemia brain damageSemantic proximity Read ↗

Validation of Altered Umbilical Cord Blood MicroRNA Expression in Neonatal Hypoxic-Ischemic EncephalopathySemantic proximity Read ↗

Genome-wide expression in neonatal encephalopathySemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.