Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Aberrant expression of semaphorin 6B affects cell phenotypes in thyroid carcinoma by activating the Notch signalling pathway.

Endokrynologia Polska - 1 Jan 2021

Lv Xiu-Juan, Chen Xin, Wang Yan, Yu Shan, Pang Lin, Huang Chao

Abstract excerpt

INTRODUCTION: Numerous semaphorins have been widely clarified to be involved in the development of multiple cancers. However, semaphorin 6B (SEMA6B) has not yet been extensively reported in cancers, especially in thyroid carcinoma. MATERIAL AND METHODS: Thyroid carcinoma RNA-Seq dataset from the TCGA database was used to assess the expression of SEMA6B in tissues, as well as its clinical significance. We adopted...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cell Line, Tumor
Share to Gene Expression Regulation, Neoplastic

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Neoplasm Invasiveness

Share to Phenotype

Share to Receptors, Notch

Share to Semaphorins

Share to Signal Transduction

Share to Thyroid Neoplasms

Source

Identifiers

PMID: 33125690
DOI: 10.5603/EP.a2020.0072

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 6 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Aberrant expression of semaphorin 6B affects cell phenotypes in thyroid carcinoma by activating the Notch signalling pathway.PMID 33125690Read ↗

Clinical significance, functional role and correlation with SEMA3B of long non-coding RNA SEMA3B-AS1 in gastric adenocarcinomaSemantic proximity Read ↗

SIX1 Activates STAT3 Signaling to Promote the Proliferation of Thyroid Carcinoma via EYA1Semantic proximity Read ↗

The association between S100A13 and HMGA1 in the modulation of thyroid cancer proliferation and invasionSemantic proximity Read ↗

Analysis of SEMA3B methylation and expression patterns in gastric cancer tissue and cell linesSemantic proximity Read ↗

Downregulation of CSN 6 attenuates papillary thyroid carcinoma progression by reducing Wnt/ β ‐catenin signaling and sensitizes cancer cells to FH 535 therapySemantic proximity Read ↗

NECTIN4 promotes papillary thyroid cancer cell proliferation, migration, and invasion and triggers EMT by activating AKTSemantic proximity Read ↗

CST6 promotes the aggressive phenotype of thyroid cancer through maintaining autophagySemantic proximity Read ↗

SEMA6C: a novel adhesion-independent FAK and YAP activator, required for cancer cell viability and growthSemantic proximity Read ↗

EpCAM silencing suppresses aggressive phenotypes and induces partial redifferentiation in anaplastic thyroid cancer cells.Semantic proximity Read ↗

The emerging role of class-3 semaphorins and their neuropilin receptors in oncologySemantic proximity Read ↗

GATA6‑induced FN1 activation promotes the proliferation, invasion and migration of oral squamous cell carcinoma cellsSemantic proximity Read ↗

SGSM2 inhibits thyroid cancer progression by activating RAP1 and enhancing competitive RAS inhibitionSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.