Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

LncRNA Xist Contributes to Endogenous Neurological Repair After Chronic Compressive Spinal Cord Injury by Promoting Angiogenesis Through the miR-32-5p/Notch-1 Axis

6 Aug 2020

Abstract excerpt

Endogenous repair after chronic compressive spinal cord injury (CCSCI) is of great clinical interest. Ischemia−hypoxia-induced angiogenesis has been proposed to play an important role during this repair process. Emerging evidence indicates that long non-coding RNAs (lncRNAs) are involved in the pathophysiological processes of various diseases. Here, we identified a lncRNA (Xist; X-inactive specific transcript)...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cancer-related molecular mechanisms research
Share to Spinal Cord Injury Research
Share to Transplantation: Methods and Outcomes

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Source

Identifiers

PMID: 32850853
DOI: 10.3389/fcell.2020.00744

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 9 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

LncRNA Xist Contributes to Endogenous Neurological Repair After Chronic Compressive Spinal Cord Injury by Promoting Angiogenesis Through the miR-32-5p/Notch-1 AxisPMID 32850853Read ↗

Epigenetic modifications of inflammation in spinal cord injurySemantic proximity Read ↗

LncRNA XIST: A Breakthrough in Inflammation-related DiseasesSemantic proximity Read ↗

Comprehensive analysis of m6A methylation modification in chronic spinal cord injury in miceSemantic proximity Read ↗

Gene Expression Profiling Analysis to Investigate the Role of CIMT after Stroke in Neurological BehaviorsSemantic proximity Read ↗

Regenerative Potential of Injured Spinal Cord in the Light of Epigenetic Regulation and ModulationSemantic proximity Read ↗

Regulation of partial endothelial-to-mesenchymal transition by circATXN1 in ischemic diseasesSemantic proximity Read ↗

RNA-seq Identifies FOS as a Potential Therapeutic Target Activating Neural Stem Cells via miR-21/SOX2 Pathway in Hypertensive Intracerebral HemorrhageSemantic proximity Read ↗

miR-328a-3p stimulates endothelial cell migration and angiogenesisSemantic proximity Read ↗

Remote limb ischemic preconditioning alleviated spinal cord injury through inhibiting proinflammatory immune response and promoting neural regenerationSemantic proximity Read ↗

Transcriptomics of Post-Stroke Angiogenesis in the Aged BrainSemantic proximity Read ↗

Research Progress of Long Non-coding RNAs in Spinal Cord InjurySemantic proximity Read ↗

Molecular insights of the injured lesions of rat spinal cords: Inflammation, apoptosis, and cell survivalSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.