Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Nucleocytoplasmic Proteomic Analysis Uncovers eRF1 and Nonsense-Mediated Decay as Modifiers of ALS/FTD C9orf72 Toxicity

13 Feb 2020

Abstract excerpt

No abstract is available from the source.

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Amyotrophic Lateral Sclerosis Research
Share to Genetic Neurodegenerative Diseases
Share to Mitochondrial Function and Pathology

Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 32059759
DOI: 10.1016/j.neuron.2020.01.020

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 5 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Nucleocytoplasmic Proteomic Analysis Uncovers eRF1 and Nonsense-Mediated Decay as Modifiers of ALS/FTD C9orf72 ToxicityPMID 32059759Read ↗

Nucleocytoplasmic Proteomic Analysis Uncovers eRF1 and Nonsense Mediated Decay as Modifiers of ALS <i>C9orf72</i> ToxicitySemantic proximity Read ↗

Modifiers of C9orf72 dipeptide repeat toxicity connect nucleocytoplasmic transport defects to FTD/ALSSemantic proximity Read ↗

A C9orf72 ALS/FTD Ortholog Acts in Endolysosomal Degradation and Lysosomal HomeostasisSemantic proximity Read ↗

ADAR2 mislocalization and widespread RNA editing aberrations in C9orf72-mediated ALS/FTDSemantic proximity Read ↗

RNA Toxicity from the ALS/FTD C9ORF72 Expansion Is Mitigated by Antisense InterventionSemantic proximity Read ↗

C9orf72-mediated ALS and FTD: multiple pathways to diseaseSemantic proximity Read ↗

Development of Therapeutics for C9ORF72 ALS/FTD-Related DisordersSemantic proximity Read ↗

Differential Toxicity of Nuclear RNA Foci versus Dipeptide Repeat Proteins in a Drosophila Model of C9ORF72 FTD/ALSSemantic proximity Read ↗

C9orf72-associated neurodegeneration in ALS-FTD: breaking new ground in ribosomal RNA and nucleolar dysfunctionSemantic proximity Read ↗

Insights into C9ORF72-Related ALS/FTD from Drosophila and iPSC ModelsSemantic proximity Read ↗

Reduced C9ORF72 function exacerbates gain of toxicity from ALS/FTD-causing repeat expansion in C9orf72Semantic proximity Read ↗

Gain of Toxicity from ALS/FTD-Linked Repeat Expansions in C9ORF72 Is Alleviated by Antisense Oligonucleotides Targeting GGGGCC-Containing RNAsSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.