Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Fungal Symbionts Produce Prostaglandin E2 to Promote Their Intestinal Colonization.

Frontiers in cellular and infection microbiology - 1 Jan 2019

Tan Tze Guan, Lim Ying Shiang, Tan Alrina, Leong Royston, Pavelka Norman

Abstract excerpt

Candida albicans is a ubiquitous fungal symbiont that resides on diverse human barrier surfaces. Both mammalian and fungal cells can convert arachidonic acid into the lipid mediator, prostaglandin E2 (PGE2), but the physiological significance of fungus-derived PGE2 remains elusive. Here we report that a C. albicans mutant deficient in PGE2 production suffered a loss of competitive fitness in the murine...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Candida albicans
Share to Dinoprostone
Share to Fungi

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to Host-Pathogen Interactions

Share to Intestinal Mucosa

Share to Macrophages

Share to Phagocytes

Share to Symbiosis

Source

Open in PubMed Open full text

Identifiers

PMID: 31681635
DOI: 10.3389/fcimb.2019.00359
PMCID: PMC6813641

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 11 closest

Fungal Symbionts Produce Prostaglandin E2 to Promote Their Intestinal Colonization.PMID 31681635Read ↗

Candida albicans Interactions with Mucosal Surfaces during Health and DiseaseSemantic proximity Read ↗

Murine Models of Candida Gastrointestinal Colonization and DisseminationSemantic proximity Read ↗

Escherichia coli, but Not Staphylococcus aureus, Functions as a Chelating Agent That Exhibits Antifungal Activity against the Pathogenic Yeast Candida albicansSemantic proximity Read ↗

Candida spp. and phagocytosis: multiple evasion mechanismsSemantic proximity Read ↗

β-glucan Exposure on the Fungal Cell Wall Tightly Correlates with Competitive Fitness of Candida Species in the Mouse Gastrointestinal TractSemantic proximity Read ↗

<i>Candida albicans</i> Colonization Modulates Murine Ethanol Consumption and Behavioral Responses Through Elevation of Serum Prostaglandin E <sub>2</sub> and Impact on the Striatal Dopamine SystemSemantic proximity Read ↗

Mutations in alternative carbon utilization pathways in Candida albicans attenuate virulence and confer pleiotropic phenotypes.Semantic proximity Read ↗

15-keto-prostaglandin E <sub>2</sub> activates host peroxisome proliferator-activated receptor gamma (PPAR-γ) to promote <i>Cryptococcus neoformans</i> growth during infectionSemantic proximity Read ↗

Estradiol impairs the Th17 immune response against Candida albicansSemantic proximity Read ↗

Augmented Enterocyte Damage During Candida albicans and Proteus mirabilis CoinfectionSemantic proximity Read ↗

Self-Regulation of Candida albicans Population Size during GI ColonizationSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.