Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Prenatal Malnutrition-Induced Epigenetic Dysregulation as a Risk Factor for Type 2 Diabetes

28 Feb 2019

Abstract excerpt

Type 2 diabetes (T2D) is commonly regarded as a disease originating from lifestyle-related factors and typically occurring after the age of 40. There is, however, consistent experimental and epidemiological data evidencing that the risk for developing T2D may largely depend on conditions early in life. In particular, intrauterine growth restriction (IUGR) induced by poor or unbalanced nutrient intake can impair...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Birth, Development, and Health
Share to Epigenetics and DNA Methylation
Share to Gestational Diabetes Research and Management

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 30944826
DOI: 10.1155/2019/3821409

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 6 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Prenatal Malnutrition-Induced Epigenetic Dysregulation as a Risk Factor for Type 2 DiabetesPMID 30944826Read ↗

Epigenetics and life-long consequences of an adverse nutritional and diabetic intrauterine environment.Semantic proximity Read ↗

Epigenetic programming of obesity and diabetes by in utero exposure to gestational diabetes mellitusSemantic proximity Read ↗

Gestational diabetes mellitus, programing and epigeneticsSemantic proximity Read ↗

Transgenerational epigenetic inheritance of diabetes risk as a consequence of early nutritional imbalancesSemantic proximity Read ↗

Nutrigenetics, epigenetics and gestational diabetes: consequences in mother and childSemantic proximity Read ↗

DNA methylation: the pivotal interaction between early-life nutrition and glucose metabolism in later lifeSemantic proximity Read ↗

Genetic, nongenetic and epigenetic risk determinants in developmental programming of type 2 diabetes.Semantic proximity Read ↗

Intrauterine programming of obesity and type 2 diabetesSemantic proximity Read ↗

Early-life Programming of Type 2 Diabetes Mellitus: Understanding the Association between Epigenetics/Genetics and Environmental FactorsSemantic proximity Read ↗

Fetal growth and developmental programmingSemantic proximity Read ↗

Aberrant DNA Methylation Mediates the Transgenerational Risk of Metabolic and Chronic Disease Due to Maternal Obesity and OvernutritionSemantic proximity Read ↗

Lifetime Exposure Model of Type 2 DiabetesSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.