Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Cellular phenotypes as inflammatory mediators in Parkinson's disease: Interventional targets and role of natural products.

Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie - 1 Oct 2018

Jiang Xu, Ganesan Palanivel, Rengarajan Thamaraiselvan, Choi Dong-Kug, Arulselvan Palanisamy

Abstract excerpt

Pathogenesis of Parkinson's disease (PD) is undoubtedly a multifactorial phenomenon, with diverse etiological agents. Pro-inflammatory mediators act as a skew that directs disease progression during neurodegenerative diseases. Understanding the dynamics of inflammation and inflammatory mediators in preventing or reducing disease progression has recently gained much attention. Inflammatory neuro-degeneration is...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Anti-Inflammatory Agents
Share to Antiparkinson Agents

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Biological Products

Share to Drug Discovery

Share to Inflammation Mediators

Share to Molecular Targeted Therapy

Share to Neuroglia

Share to Parkinson Disease

Source

Identifiers

PMID: 30119171
DOI: 10.1016/j.biopha.2018.06.162

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Cellular phenotypes as inflammatory mediators in Parkinson's disease: Interventional targets and role of natural products.PMID 30119171Read ↗

Microglial phenotypes in Parkinson's disease and animal models of the disease.Semantic proximity Read ↗

Cellular and Molecular Mechanisms of Parkinson’s Disease: Neurotoxins, Causative Genes, and Inflammatory CytokinesSemantic proximity Read ↗

Emerging therapeutic approaches targeting neuroinflammation in neurodegenerative diseasesSemantic proximity Read ↗

Deciphering variability in the role of interleukin-1β in Parkinson’s diseaseSemantic proximity Read ↗

Pharmacological Modulation of Functional Phenotypes of Microglia in Neurodegenerative DiseasesSemantic proximity Read ↗

Pathogenesis of Parkinson’s disease: oxidative stress, environmental impact factors and inflammatory processesSemantic proximity Read ↗

Neuroinflammation in Parkinson's disease: role in neurodegeneration and tissue repair.Semantic proximity Read ↗

Immunoregulation of Microglia M2 polarization is an unrecognized physiological function of α -SynucleinSemantic proximity Read ↗

PARK7/DJ-1 in microglia: implications in Parkinson’s disease and relevance as a therapeutic targetSemantic proximity Read ↗

Neuroinflammation and Autophagy in Parkinson’s Disease—Novel PerspectivesSemantic proximity Read ↗

The role of innate and adaptive immunity in Parkinson's disease.Semantic proximity Read ↗

IGF2-reprogrammed macrophages ameliorate the inflammatory response and protects against the neurodegenerative and neuroinflammatory process in Parkinson`s disease models.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.