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Article

Asian G6PD-Mahidol Reticulocytes Sustain Normal Plasmodium Vivax Development.

The Journal of infectious diseases - 15 Jul 2017

Bancone Germana, Malleret Benoit, Suwanarusk Rossarin, Chowwiwat Nongnud, Chu Cindy S, McGready Rose, Rénia Laurent, Nosten François, Russell Bruce

Abstract excerpt

Glucose-6-phosphate dehydrogenase (G6PD) deficiency is the most common enzymatic disorder in humans and appears to be protective against falciparum severe malaria. Controversially, it is also thought that Plasmodium vivax has driven the recent selection of G6PD alleles. We use an experimental approach to determine whether G6PD-MahidolG487A variant, a widespread cause of severe G6PD deficiency in Southeast Asia,...

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Identifiers

PMID: 28591790
DOI: 10.1093/infdis/jix278
PMCID: PMC5853331

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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