Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Sinorhizobium fredii HH103 Invades Lotus burttii by Crack Entry in a Nod Factor-and Surface Polysaccharide-Dependent Manner.

Molecular plant-microbe interactions : MPMI - 1 Dec 2016

Acosta-Jurado Sebastián, Rodríguez-Navarro Dulce-Nombre, Kawaharada Yasuyuki, Perea Juan Fernández, Gil-Serrano Antonio, Jin Haojie, An Qi, Rodríguez-Carvajal Miguel A, Andersen Stig U, Sandal Niels, Stougaard Jens, Vinardell José-María, Ruiz-Sainz José E

Abstract excerpt

Sinorhizobium fredii HH103-Rifr, a broad host range rhizobial strain, induces nitrogen-fixing nodules in Lotus burttii but ineffective nodules in L. japonicus. Confocal microscopy studies showed that Mesorhizobium loti MAFF303099 and S. fredii HH103-Rifr invade L. burttii roots through infection threads or epidermal cracks, respectively. Infection threads in root hairs were not observed in L. burttii plants...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Host Specificity
Share to Lotus

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Plant Root Nodulation

Share to Plant Roots

Share to Polysaccharides, Bacterial

Share to Purines

Share to Pyrimidines

Share to Symbiosis

Source

Identifiers

PMID: 27827003
DOI: 10.1094/MPMI-09-16-0195-R

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 5 Sept 2026. Manifest publication-graph-cohort-2026-09-05.1.

Source receipt: pubmed, retrieved 5 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Sinorhizobium fredii HH103 Invades Lotus burttii by Crack Entry in a Nod Factor-and Surface Polysaccharide-Dependent Manner.PMID 27827003Read ↗

Sinorhizobium fredii HH103 syrM inactivation affects the expression of a large number of genes, impairs nodulation with soybean and extends the host-range to Lotus japonicus.Semantic proximity Read ↗

The molecular network governing nodule organogenesis and infection in the model legume Lotus japonicus.Semantic proximity Read ↗

Nodulation gene mutants of Mesorhizobium loti R7A-nodZ and nolL mutants have host-specific phenotypes on Lotus spp.Semantic proximity Read ↗

Polymorphic infection and organogenesis patterns induced by a Rhizobium leguminosarum isolate from Lotus root nodules are determined by the host genotype.Semantic proximity Read ↗

Nod factor signaling genes and their function in the early stages of Rhizobium infection.Semantic proximity Read ↗

Single-cell analysis identifies genes facilitating rhizobium infection in Lotus japonicus.Semantic proximity Read ↗

Improved characterization of nod factors and genetically based variation in LysM Receptor domains identify amino acids expendable for nod factor recognition in Lotus spp.Semantic proximity Read ↗

The Sinorhizobium fredii HH103 lipopolysaccharide is not only relevant at early soybean nodulation stages but also for symbiosome stability in mature nodules.Semantic proximity Read ↗

Reactions of Lotus japonicus ecotypes and mutants to root parasitic plants.Semantic proximity Read ↗

Structure and biological roles of Sinorhizobium fredii HH103 exopolysaccharide.Semantic proximity Read ↗

Mutation in GDP-fucose synthesis genes of Sinorhizobium fredii alters Nod factors and significantly decreases competitiveness to nodulate soybeans.Semantic proximity Read ↗

Rhizobium meliloti lipooligosaccharide nodulation factors: different structural requirements for bacterial entry into target root hair cells and induction of plant symbiotic developmental responses.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.