Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Chronic MeHg exposure modifies the histone H3K4me3 epigenetic landscape in Caenorhabditis elegans

9 Oct 2016

Abstract excerpt

Methylmercury (MeHg) is a persistent environmental pollutant that occurs in the food chain, at occupational sites, and via medical procedures. Exposure in humans and animal models results in renal, neuro, and reproductive toxicities. In this study, we demonstrate that chronic exposure to MeHg (10μM) causes epigenetic landscape modifications of histone H3K4 trimethylation (H3K4me3) marks in Caenorhabditis elegans...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Birth, Development, and Health
Share to Genetics, Aging, and Longevity in Model Organisms
Share to Health, Environment, Cognitive Aging

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 27717699
DOI: 10.1016/j.cbpc.2016.10.001

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 6 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Chronic MeHg exposure modifies the histone H3K4me3 epigenetic landscape in Caenorhabditis elegansPMID 27717699Read ↗

Methylmercury exposure increases lipocalin related (lpr) and decreases activated in blocked unfolded protein response (abu) genes and specific miRNAs in Caenorhabditis elegans.Semantic proximity Read ↗

Transcriptomic, proteomic, and metabolomic analyses identify candidate pathways linking maternal cadmium exposure to altered neurodevelopment and behaviorSemantic proximity Read ↗

Methylmercury Induces Metabolic Alterations in Caenorhabditis elegans: Role for C/EBP Transcription Factor.Semantic proximity Read ↗

Swimming Into View: Zebrafish Uncover Targets, Mechanisms, and Therapies for Cadmium ToxicitySemantic proximity Read ↗

The Memory of Environmental Chemical Exposure in C. elegans Is Dependent on the Jumonji Demethylases jmjd-2 and jmjd-3/utx-1Semantic proximity Read ↗

The nervous system is the major target for Gold nanoparticles: Evidence from RNA sequencing data of <i>C. elegans</i>Semantic proximity Read ↗

Caenorhabditis Elegans as a Model for Environmental EpigeneticsSemantic proximity Read ↗

Genetic and epigenetic interplay allows rapid transgenerational adaptation to metal pollution in zebrafishSemantic proximity Read ↗

Cadmium-induced reproductive toxicity combined with a correlation to the oogenesis process and competing endogenous RNA networks based on a Caenorhabditis elegans modelSemantic proximity Read ↗

Toxicogenomic analysis of Caenorhabditis elegans reveals novel genes and pathways involved in the resistance to cadmium toxicitySemantic proximity Read ↗

Testicular Dysgenesis Syndrome and Long-Lasting Epigenetic Silencing of Mouse Sperm Genes Involved in the Reproductive System after Prenatal Exposure to DEHPSemantic proximity Read ↗

Hexavalent chromium neurotoxicity in Caenorhabditis elegans targeted GABAergic, cholinergic, and dopaminergic neurons, and contributed to an aged phenotype.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.