Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

That's not it, either-neither polymorphisms in PHOX2B nor in MIF are involved in sudden infant death syndrome (SIDS).

International journal of legal medicine - 1 Sept 2015

Poetsch Micaela, Todt Rebecca, Vennemann Mechtild, Bajanowski Thomas

Abstract excerpt

The occurrence of sudden infant death syndrome (SIDS) has been linked to several genetic risk factors, e.g. genes involved in the neuroadrenergic system, variations in serotonin reporter genes or mutations in long-QT syndrome genes. Additionally, polymorphisms in genes with impact in sleep disorder syndromes have been proposed to be of importance as genetic risk factors for SIDS. In this study, we investigated...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Infant, Newborn
Share to Homeodomain Proteins

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to Infant

Share to Intramolecular Oxidoreductases

Share to Macrophage Migration-Inhibitory Factors

Share to Promoter Regions, Genetic

Share to Sudden Infant Death

Share to Transcription Factors

Source

Identifiers

PMID: 26104808
DOI: 10.1007/s00414-015-1213-3

Genes

MIF

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

That's not it, either-neither polymorphisms in PHOX2B nor in MIF are involved in sudden infant death syndrome (SIDS).PMID 26104808Read ↗

PHOX2B polyalanine repeat length is associated with sudden infant death syndrome and unclassified sudden infant death in the Dutch population.Semantic proximity Read ↗

Gene variants predisposing to SIDS: current knowledge.Semantic proximity Read ↗

A Common 3′UTR Variant of the PHOX2B Gene Is Associated With Infant Life-Threatening and Sudden Death Events in the Italian PopulationSemantic proximity Read ↗

Sudden infant death syndrome (SIDS) and polymorphisms in Monoamine oxidase A gene (MAOA): a revisit.Semantic proximity Read ↗

Non-polyalanine repeat mutation in PHOX2B is detected in autopsy cases of sudden unexpected infant death.Semantic proximity Read ↗

Genomic risk factors in sudden infant death syndromeSemantic proximity Read ↗

Sudden infant death syndrome revisited: serotonin transporter gene, polymorphisms and promoter methylation.Semantic proximity Read ↗

HTR2A variation and sudden infant death syndrome: a case-control analysis.Semantic proximity Read ↗

SNP association and sequence analysis of the NOS1AP gene in SIDS.Semantic proximity Read ↗

Association between monoamine oxidase A promoter polymorphism and the risk of sudden infant death syndrome: a meta-analysis.Semantic proximity Read ↗

Association between a functional polymorphism in the MAOA gene and sudden infant death syndrome.Semantic proximity Read ↗

Sudden infant death syndrome: another year of new hope but no cure.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.