Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Chronic Toxoplasma gondii in Nurr1-null heterozygous mice exacerbates elevated open field activity.

PloS one - 1 Jan 2015

Eells Jeffrey B, Varela-Stokes Andrea, Guo-Ross Shirley X, Kummari Evangel, Smith Holly M, Cox Arin D, Lindsay David S

Abstract excerpt

Latent infection with Toxoplasma gondii is common in humans (approximately 30% of the global population) and is a significant risk factor for schizophrenia. Since prevalence of T. gondii infection is far greater than prevalence of schizophrenia (0.5-1%), genetic risk factors are likely also neces...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Antigens, Protozoan
Share to Anxiety
Share to Avoidance Learning

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Nuclear Receptor Subfamily 4, Group A, Member 2

Share to Reflex, Startle

Share to Sensory Gating

Share to Toxoplasma

Share to Toxoplasmosis

Source

Open in PubMed Open full text

Identifiers

PMID: 25855987
DOI: 10.1371/journal.pone.0119280
PMCID: PMC4391871

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 4 Sept 2026. Prepared record publication-25855987-2026-09-04.2. Manifest publication-graph-cohort-2026-09-04.2.

Source receipt: PubMed via Helena Literature Corpus, retrieved 4 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Chronic Toxoplasma gondii in Nurr1-null heterozygous mice exacerbates elevated open field activity.PMID 25855987Read ↗

<i>Toxoplasma</i> infection alters dopamine-sensitive behaviors and host gene expression patterns associated with neuropsychiatric diseaseSemantic proximity Read ↗

Epigenetic Manipulation of Psychiatric Behavioral Disorders Induced by Toxoplasma gondiiSemantic proximity Read ↗

Toxoplasmosis and Psychiatric and Neurological Disorders: A Step toward Understanding Parasite PathogenesisSemantic proximity Read ↗

Effects of latent infection of Toxoplasma gondii strains with different genotypes on mouse behavior and brain transcriptsSemantic proximity Read ↗

The known and missing links between Toxoplasma gondii and schizophreniaSemantic proximity Read ↗

<i>Toxoplasma gondii</i>‐induced neuronal alterationsSemantic proximity Read ↗

A metabolite attenuates neuroinflammation, synaptic loss and cognitive deficits induced by chronic infection of Toxoplasma gondiiSemantic proximity Read ↗

The effect of Toxoplasma gondii on host behaviour : studies on evolution and mechanisms of actionSemantic proximity Read ↗

Toxoplasma gondii-altered host behaviour: elucidating the role of parasite-produced dopamineSemantic proximity Read ↗

Altered motor activity, exploration and anxiety in heterozygous neuregulin 1 mutant mice: implications for understanding schizophrenia.Semantic proximity Read ↗

Exploratory and habituation phenotype of heterozygous and homozygous COMT knockout mice.Semantic proximity Read ↗

Behavioral phenotypes in schizophrenic animal models with multiple combinations of genetic and environmental factors.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.