Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Transcriptional Factor DLX3 Promotes the Gene Expression of Enamel Matrix Proteins during Amelogenesis

27 Mar 2015

Abstract excerpt

Mutation of distal-less homeobox 3 (DLX3) is responsible for human tricho-dento-osseous syndrome (TDO) with amelogenesis imperfecta, indicating a crucial role of DLX3 in amelogenesis. However, the expression pattern of DLX3 and its specific function in amelogenesis remain largely unknown. The aim of this study was to investigate the effects of DLX3 on enamel matrix protein (EMP) genes. By immunohistochemistry...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Bone and Dental Protein Studies
Share to dental development and anomalies
Share to Periodontal Regeneration and Treatments

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 25815730
DOI: 10.1371/journal.pone.0121288

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 5 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Transcriptional Factor DLX3 Promotes the Gene Expression of Enamel Matrix Proteins during AmelogenesisPMID 25815730Read ↗

Unraveling the roles of <i>Mast4</i> in amelogenesis via regulating DLX3 and stem cell maintenance of mouse incisorsSemantic proximity Read ↗

Regulation and function of Dlx3 in vertebrate development.Semantic proximity Read ↗

A novel DLX3 mutation causes tricho-dento-osseous syndrome with abnormal enamel structure and formation.Semantic proximity Read ↗

miR-675 promotes odontogenic differentiation of human dental pulp cells by epigenetic regulation of DLX3Semantic proximity Read ↗

Differential Effects of DLX3 Mutations Drive Phenotypic Variability in Tricho-Dento-Osseous Syndrome via Direct Activation of WNT10A.Semantic proximity Read ↗

DLX3 mutation negatively regulates odontogenic differentiation of human dental pulp cells.Semantic proximity Read ↗

Identification of a mutation in DLX3 associated with tricho-dento- osseous (TDO) syndromeSemantic proximity Read ↗

Dlx3 is a crucial regulator of hair follicle differentiation and cyclingSemantic proximity Read ↗

Integrated Genetic Networks and Epigenetic Regulation in Tooth Development and MaturationSemantic proximity Read ↗

Homeobox gene Dlx3 is regulated by p63 during ectoderm development: relevance in the pathogenesis of ectodermal dysplasias.Semantic proximity Read ↗

A model for the molecular underpinnings of tooth defects in Axenfeld–Rieger syndromeSemantic proximity Read ↗

Unexpected identification of a recurrent mutation in the DLX3 gene causing amelogenesis imperfecta.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.