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Article

Deciphering the role of phosphatidylinositol 3-kinase mutations in human epidermal growth factor receptor 2-positive breast cancer.

Journal of clinical oncology : official journal of the American Society of Clinical Oncology - 20 Apr 2015

Goel Shom, Krop Ian E

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Share to Antibodies, Monoclonal, Humanized
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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

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Identifiers

PMID: 25800754
DOI: 10.1200/JCO.2014.60.0742

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Deciphering the role of phosphatidylinositol 3-kinase mutations in human epidermal growth factor receptor 2-positive breast cancer.PMID 25800754Read ↗

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Assessment of somatic mutations in phosphatidylinositol 3-kinase gene in human lymphoma and acute leukaemia.Semantic proximity Read ↗

Phosphatidylinositol 3‐kinase pathway genomic alterations in 60,991 diverse solid tumors informs targeted therapy opportunitiesSemantic proximity Read ↗

PIK3CA mutations correlate with hormone receptors, node metastasis, and ERBB2, and are mutually exclusive with PTEN loss in human breast carcinoma.Semantic proximity Read ↗

Targeting PIK3CA Alterations in Hormone Receptor-Positive, Human Epidermal Growth Factor Receptor-2-Negative Advanced Breast Cancer: New Therapeutic Approaches and Practical Considerations.Semantic proximity Read ↗

Cancer-specific mutations in phosphatidylinositol 3-kinase.Semantic proximity Read ↗

Phosphoinositide 3-kinase mutations in breast cancer: a "good" activating mutation?Semantic proximity Read ↗

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