Article
A truncated NLR protein, TIR-NBS2, is required for activated defense responses in the exo70B1 mutant.
PLoS genetics - 1 Jan 2015
Zhao Ting, Rui Lu, Li Juan, Nishimura Marc T, Vogel John P, Liu Na, Liu Simu, Zhao Yaofei, Dangl Jeffery L, Tang Dingzhong
Abstract excerpt
During exocytosis, the evolutionarily conserved exocyst complex tethers Golgi-derived vesicles to the target plasma membrane, a critical function for secretory pathways. Here we show that exo70B1 loss-of-function mutants express activated defense responses upon infection and express enhanced resistance to fungal, oomycete and bacterial pathogens. In a screen for mutants that suppress exo70B1 resistance, we...
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