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Article

Mechanisms in endocrinology: autoimmune thyroid disease: old and new players.

European journal of endocrinology - 1 Jun 2014

Effraimidis Grigoris, Wiersinga Wilmar M

Abstract excerpt

The last 10 years have seen some progress in understanding the etiology of autoimmune thyroid disease (AITD). The female preponderance can now be explained - at least in part - by fetal microchimerism and X-chromosome inactivation. The number of identified susceptibility genes for AITD is increasing (among others now including TSHR, TG, HLA, CTLA4, PTPN22, CD40, FCRL3, IL2RA, and FOXP3), but these genes together...

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Topics

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

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Share to Stress, Psychological

Share to Thyroiditis, Autoimmune

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Source

Identifiers

PMID: 24609834
DOI: 10.1530/EJE-14-0047

Genes

CTLA4
FCRL3
FOXP3
PTPN22
TSHR

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 6 Sept 2026. Manifest publication-graph-cohort-2026-09-06.1.

Source receipt: pubmed, retrieved 6 Sept 2026.

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