Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

KIR gene content does not contribute to susceptibility to Graves' disease.

Iranian journal of immunology : IJI - 1 Sept 2013

Ashouri Elham, Dabbaghmanesh Mohammad Hossein, Hadaegh Amirhossein, Rowhanirad Soodeh, Bakhshayashkaram Marizeh, Omrani Gholamhossein Ranjbar

Abstract excerpt

BACKGROUND: Killer cell immunoglobulin-like receptors (KIR) are expressed on NK cells and a subset of T cells. The variable KIR receptors along with their ligands, HLA class I, influence risk for autoimmune and malignant diseases. OBJECTIVE: To investigate the KIR gene profiles in relation to susceptibility to Graves' disease in patients with ophthalmopathy. METHODS: KIR genes profiles were analyzed in 90...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to DNA Mutational Analysis
Share to Gene Expression Regulation

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to Genetic Association Studies

Share to Graves Disease

Share to Graves Ophthalmopathy

Share to Killer Cells, Natural

Share to Receptors, KIR

Share to Transcriptome

Source

Identifiers

PMID: 24076592
DOI: 10.22034/iji.2013.16824

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

KIR gene content does not contribute to susceptibility to Graves' disease.PMID 24076592Read ↗

[Genotype analysis of killer cell immunoglobulin-like receptors in Graves' disease patients].Semantic proximity Read ↗

Susceptibility genes in Graves' ophthalmopathy: searching for a needle in a haystack?Semantic proximity Read ↗

Genetic profiling in Graves' disease: further evidence for lack of a distinct genetic contribution to Graves' ophthalmopathy.Semantic proximity Read ↗

[Correlation between gene polymorphisms of killer cell immunoglobulin-like receptors and their ligands and Graves' disease].Semantic proximity Read ↗

MHC class II region, CTLA4 gene, and ophthalmopathy in patients with Graves' diseaseSemantic proximity Read ↗

HLA-DRB1*03 is a susceptibility gene in patients with Graves' disease with and without ophthalmopathy.Semantic proximity Read ↗

Lack of association of interleukin-18 gene polymorphisms with susceptibility of Japanese populations to Graves' disease or Graves' ophthalmopathy.Semantic proximity Read ↗

HLA DQA1*0501 and DRB1*0301 antigens do not independently convey susceptibility to Graves' disease.Semantic proximity Read ↗

Limited genetic susceptibility to severe Graves' ophthalmopathy: no role for CTLA-4 but evidence for an environmental etiology.Semantic proximity Read ↗

Killer immunoglobulin-like receptor genes in uveitis.Semantic proximity Read ↗

Lack of an association between alleles of interleukin-1 alpha and interleukin-1 receptor antagonist genes and Graves' disease in a North American Caucasian population.Semantic proximity Read ↗

Lack of association of Graves' disease with the A2 allele of the interleukin-1 receptor antagonist gene in a white European population.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.