Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Suppression of HPV-16 late L1 5′-splice site SD3632 by binding of hnRNP D proteins and hnRNP A2/B1 to upstream AUAGUA RNA motifs

5 Sept 2013

Abstract excerpt

Human papillomavirus type 16 (HPV-16) 5'-splice site SD3632 is used exclusively to produce late L1 mRNAs. We identified a 34-nt splicing inhibitory element located immediately upstream of HPV-16 late 5'-splice site SD3632. Two AUAGUA motifs located in these 34 nt inhibited SD3632. Two nucleotide substitutions in each of the HPV-16 specific AUAGUA motifs alleviated splicing inhibition and induced late L1 mRNA...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Herpesvirus Infections and Treatments
Share to Molecular Biology Techniques and Applications
Share to T-cell and Retrovirus Studies

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 24013563
DOI: 10.1093/nar/gkt803

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 6 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Suppression of HPV-16 late L1 5′-splice site SD3632 by binding of hnRNP D proteins and hnRNP A2/B1 to upstream AUAGUA RNA motifsPMID 24013563Read ↗

A Downstream Polyadenylation Element in Human Papillomavirus Type 16 L2 Encodes Multiple GGG Motifs and Interacts with hnRNP HSemantic proximity Read ↗

A splicing enhancer in the E4 coding region of human papillomavirus type 16 is required for early mRNA splicing and polyadenylation as well as inhibition of premature late gene expression.Semantic proximity Read ↗

HPV-16 RNA processingSemantic proximity Read ↗

hnRNP G/RBMX enhances HPV16 E2 mRNA splicing through a novel splicing enhancer and inhibits production of spliced E7 oncogene mRNAsSemantic proximity Read ↗

Analysis of novel human papillomavirus type 16 late mRNAs in differentiated W12 cervical epithelial cellsSemantic proximity Read ↗

The human papillomavirus type 16 negative regulatory RNA element interacts with three proteins that act at different posttranscriptional levelsSemantic proximity Read ↗

Heterogeneous Nuclear Ribonucleoprotein C Proteins Interact with the Human Papillomavirus Type 16 (HPV16) Early 3′-Untranslated Region and Alleviate Suppression of HPV16 Late L1 mRNA SplicingSemantic proximity Read ↗

HnRNP D activates production of HPV16 E1 and E6 mRNAs by promoting intron retentionSemantic proximity Read ↗

Alternative splicing in the genome of HPV and its regulationSemantic proximity Read ↗

The DNA damage response activates HPV16 late gene expression at the level of RNA processingSemantic proximity Read ↗

Role of the DNA Damage Response in Human Papillomavirus RNA Splicing and PolyadenylationSemantic proximity Read ↗

Identification of an hnRNP A1-Dependent Splicing Silencer in the Human Papillomavirus Type 16 L1 Coding Region That Prevents Premature Expression of the Late L1 GeneSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.