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Article

Encephalomyocarditis virus disrupts stress granules, the critical platform for triggering antiviral innate immune responses.

Journal of virology - 1 Sept 2013

Ng Chen Seng, Jogi Michihiko, Yoo Ji-Seung, Onomoto Koji, Koike Satoshi, Iwasaki Takuya, Yoneyama Mitsutoshi, Kato Hiroki, Fujita Takashi

Abstract excerpt

In response to stress, cells induce ribonucleoprotein aggregates, termed stress granules (SGs). SGs are transient loci containing translation-stalled mRNA, which is eventually degraded or recycled for translation. Infection of some viruses, including influenza A virus with a deletion of nonstructural protein 1 (IAVΔNS1), induces SG-like protein aggregates. Previously, we showed that IAVΔNS1-induced SGs are...

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Identifiers

PMID: 23785203
DOI: 10.1128/JVI.03248-12
PMCID: PMC3754122

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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