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Article

The SEPS1 G-105A polymorphism is associated with risk of spontaneous preterm birth in a Chinese population.

PloS one - 1 Jan 2013

Wang Yan, Yang Xiao, Zheng Yong, Wu Zhi-Hao, Zhang Xiao-Ai, Li Qiu-Ping, He Xi-Yu, Wang Chun-Zhi, Feng Zhi-Chun

Abstract excerpt

Inflammation plays an important role in the etiology and pathophysiology of spontaneous preterm birth (SPTB), and selenoprotein S (SEPS1) is involved in regulating the inflammatory response. Recently the G-105A promoter polymorphism in SEPS1 was shown to increase pro-inflammatory cytokine expression. We examined whether this functional polymorphism was related to the risk of SPTB in a Chinese population. We also...

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Identifiers

PMID: 23776519
DOI: 10.1371/journal.pone.0065657
PMCID: PMC3679159

Genes

SPTB

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 6 Sept 2026. Manifest publication-graph-cohort-2026-09-06.1.

Source receipt: pubmed, retrieved 6 Sept 2026.

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