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Article

Snail and Slug, key regulators of TGF-β-induced EMT, are sufficient for the induction of single-cell invasion

22 Apr 2013

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Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 23618854
DOI: 10.1016/j.bbrc.2013.04.037

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Related research

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Snail and Slug, key regulators of TGF-β-induced EMT, are sufficient for the induction of single-cell invasionPMID 23618854Read ↗

Snail and Slug Promote Epithelial-Mesenchymal Transition through β-Catenin–T-Cell Factor-4-dependent Expression of Transforming Growth Factor-β3Semantic proximity Read ↗

Multilayer control of the EMT master regulators.Semantic proximity Read ↗

The Role of Snail in EMT and TumorigenesisSemantic proximity Read ↗

A positive role for Myc in TGFβ-induced Snail transcription and epithelial-to-mesenchymal transitionSemantic proximity Read ↗

The contribution of TGF-β in Epithelial–Mesenchymal Transition (EMT): Down-regulation of E-cadherin via snailSemantic proximity Read ↗

Canonical TGFβ signaling induces collective invasion in colorectal carcinogenesis through a Snail1- and Zeb1-independent partial EMTSemantic proximity Read ↗

A SNAIL1–SMAD3/4 transcriptional repressor complex promotes TGF-β mediated epithelial–mesenchymal transitionSemantic proximity Read ↗

STAT3 integrates cooperative Ras and TGF-β signals that induce Snail expressionSemantic proximity Read ↗

Central role of Snail1 in the regulation of EMT and resistance in cancer: a target for therapeutic interventionSemantic proximity Read ↗

Reciprocal expression of Slug and Snail in human oral cancer cellsSemantic proximity Read ↗

Canonical BMP Signaling Executes Epithelial-Mesenchymal Transition Downstream of SNAIL1Semantic proximity Read ↗

Snail induction is an early response to Gli1 that determines the efficiency of epithelial transformationSemantic proximity Read ↗

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