Variants in RUNX3 contribute to susceptibility to psoriatic arthritis, exhibiting further common ground with ankylosing spondylitis.

doi:10.1002/art.37885

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Community

Back to search

Article

Variants in RUNX3 contribute to susceptibility to psoriatic arthritis, exhibiting further common ground with ankylosing spondylitis.

Arthritis and rheumatism - 1 May 2013

Apel Maria, Uebe Steffen, Bowes John, Giardina Emiliano, Korendowych Eleanor, Juneblad Kristina, Pasutto Francesca, Ekici Arif B, McManus Ross, Ho Pauline, Bruce Ian N, Ryan Anthony W, Behrens Frank, Böhm Beate, Traupe Heiko, Lohmann Jörg, Gieger Christian, Wichmann Heinz-Erich, Padyukov Leonid, Fitzgerald Oliver, Alenius Gerd-Marie, McHugh Neil J, Novelli Giuseppe, Burkhardt Harald, Barton Anne, Reis André, Hüffmeier Ulrike

Abstract excerpt

OBJECTIVE: Psoriatic arthritis (PsA) is a common inflammatory joint disease distinct from other chronic arthritides and frequently accompanied by psoriasis vulgaris. In a first genome-wide association study (GWAS), we were able to identify several genetic risk factors. However, even combined with previously identified factors, the genetic contribution to disease was not fully explained. Therefore, we undertook...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Case-Control Studies

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Cohort Studies

Share to Core Binding Factor Alpha 3 Subunit

Share to Gene Frequency

Share to Genome-Wide Association Study

Share to Linkage Disequilibrium

Share to Polymorphism, Single Nucleotide

Share to Spondylitis, Ankylosing

Source

Open in PubMed

Identifiers

PMID: 23401011
DOI: 10.1002/art.37885

Genes

RUNX3

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 6 Sept 2026. Manifest publication-graph-cohort-2026-09-06.1.

Source receipt: pubmed, retrieved 6 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Variants in RUNX3 contribute to susceptibility to psoriatic arthritis, exhibiting further common ground with ankylosing spondylitis.PMID 23401011Read ↗

Genetic overlap between psoriatic arthritis and ankylosing spondylitis in a Chinese population.Semantic proximity Read ↗

Genetic susceptibility factors for psoriatic arthritis.Semantic proximity Read ↗

What have genome-wide studies told us about psoriatic arthritis?Semantic proximity Read ↗

Genetics of psoriatic arthritis.Semantic proximity Read ↗

Insights into the pathogenesis of psoriatic arthritis from genetic studies.Semantic proximity Read ↗

Genome-wide Association Analysis of Psoriatic Arthritis and Cutaneous Psoriasis Reveals Differences in Their Genetic Architecture.Semantic proximity Read ↗

Psoriasis and Psoriatic Arthritis-Associated Genes, Cytokines, and Human Leukocyte Antigens.Semantic proximity Read ↗

Genetic Studies Investigating Susceptibility to Psoriatic Arthritis: A Narrative Review.Semantic proximity Read ↗

The genetics of psoriasis and psoriatic arthritis.Semantic proximity Read ↗

The Genetics of Psoriasis and Psoriatic Arthritis.Semantic proximity Read ↗

Common variants at TRAF3IP2 are associated with susceptibility to psoriatic arthritis and psoriasis.Semantic proximity Read ↗

Genome-wide meta-analysis of psoriatic arthritis identifies susceptibility locus at REL.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.