Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

West Nile Virus Infection Causes Endocytosis of a Specific Subset of Tight Junction Membrane Proteins

24 May 2012

Abstract excerpt

West Nile virus (WNV) is a blood-borne pathogen that causes systemic infections and serious neurological disease in human and animals. The most common route of infection is mosquito bites and therefore, the virus must cross a number of polarized cell layers to gain access to organ tissue and the central nervous system. Resistance to trans-cellular movement of macromolecules between epithelial and endothelial...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Mosquito-borne diseases and control
Share to Trace Elements in Health
Share to Vibrio bacteria research studies

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 22655077
DOI: 10.1371/journal.pone.0037886

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

West Nile Virus Infection Causes Endocytosis of a Specific Subset of Tight Junction Membrane ProteinsPMID 22655077Read ↗

West Nile Virus Capsid Degradation of Claudin Proteins Disrupts Epithelial Barrier FunctionSemantic proximity Read ↗

Tight Junctions Go Viral!Semantic proximity Read ↗

Endocytosis of flavivirus NS1 is required for NS1-mediated endothelial hyperpermeability and is abolished by a single N-glycosylation site mutationSemantic proximity Read ↗

Virus-associated disruption of mucosal epithelial tight junctions and its role in viral transmission and spreadSemantic proximity Read ↗

Alteration of cell junctions during viral infectionSemantic proximity Read ↗

Exosomes serve as novel modes of tick-borne flavivirus transmission from arthropod to human cells and facilitates dissemination of viral RNA and proteins to the vertebrate neuronal cellsSemantic proximity Read ↗

Molecular Mechanism and Role of Japanese Encephalitis Virus Infection in Central Nervous System-Mediated DiseasesSemantic proximity Read ↗

Lumpy skin disease virus enters into host cells via dynamin-mediated endocytosis and macropinocytosisSemantic proximity Read ↗

Beyond the Surface: Endocytosis of Mosquito-Borne FlavivirusesSemantic proximity Read ↗

Orthobunyavirus Entry into Neurons and Other Mammalian Cells Occurs via Clathrin-Mediated Endocytosis and Requires Trafficking into Early EndosomesSemantic proximity Read ↗

JC Polyomavirus Entry by Clathrin-Mediated Endocytosis Is Driven by β-ArrestinSemantic proximity Read ↗

Venezuelan equine encephalitis virus infection of mosquito cells requires acidification as well as mosquito homologs of the endocytic proteins Rab5 and Rab7Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.